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Cell Cycle (Georgetown, Tex.)|March 27, 2004
The significance of p16INK4a in cell defenses against transformationSarah Drayton, Sharon Brookes, Janice Rowe, et al.Cancer Research|November 15, 2011
Contrasting behavior of the p18INK4c and p16INK4a tumor suppressors in both replicative and oncogene-induced senescenceSladjana Gagrica, Sharon Brookes, Emma Anderton, et al.Experimental Cell Research|July 22, 2004
Contribution of p16(INK4a) to replicative senescence of human fibroblastsSharon Brookes, Janice Rowe, Ana Gutierrez Del Arroyo, et al.Plos One|July 25, 2014
Role of polycomb group proteins in the DNA damage response--a reassessmentHollie Chandler, Harshil Patel, Richard Palermo, et al.Aging Cell|September 30, 2015
CBX7 and miR-9 are part of an autoregulatory loop controlling p16(INK) (4a)Ana O'Loghlen, Sharon Brookes, Nadine Martin, et al.Nature Structural & Molecular Biology|June 15, 2010
Role for the MOV10 RNA helicase in polycomb-mediated repression of the INK4a tumor suppressorSelma El Messaoudi-Aubert, James Nicholls, Goedele N Maertens, et al.Cell Cycle (Georgetown, Tex.)|February 20, 2015
Evidence for a CDK4-dependent checkpoint in a conditional model of cellular senescenceSharon Brookes, Sladjana Gagrica, Elaine Sanij, et al.Molecular and Cellular Biology|April 11, 2007
CDK4 and CDK6 delay senescence by kinase-dependent and p16INK4a-independent mechanismsMargarida Ruas, Fiona Gregory, Rebecca Jones, et al.Cancer Research|October 3, 2007
A CDKN2A mutation in familial melanoma that abrogates binding of p16INK4a to CDK4 but not CDK6Rebecca Jones, Margarida Ruas, Fiona Gregory, et al.Oncogene|October 21, 2014
The nuclear receptor NR2E1/TLX controls senescenceAna O'Loghlen, Nadine Martin, Benjamin Krusche, et al.Pageof 2