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The Journal of Physiology|August 11, 2016
K<sub>V</sub> 7/M channels as targets for lipopolysaccharide-induced inflammatory neuronal hyperexcitabilityArik Tzour, Hodaya Leibovich, Omer Barkai, et al.Biochimica Et Biophysica Acta|September 15, 2016
Privileged crosstalk between TRPV1 channels and mitochondrial calcium shuttling machinery controls nociceptionIulia I Nita, Yaki Caspi, Sagi Gudes, et al.Cells|April 25, 2020
Abnormal Reinnervation of Denervated Areas Following Nerve Injury Facilitates Neuropathic PainHodaya Leibovich, Nahum Buzaglo, Shlomo Tsuriel, et al.Pain|April 2, 2019
Platelet-derived growth factor activates nociceptive neurons by inhibiting M-current and contributes to inflammatory painOmer Barkai, Stephanie Puig, Shaya Lev, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 27, 2009
Membrane lipid modulations remove divalent open channel block from TRP-like and NMDA channelsMoshe Parnas, Ben Katz, Shaya Lev, et al.Science Advances|June 20, 2025
Opposite regulation of medullary pain-related projection neuron excitability in acute and chronic painBen Title, Enrique Velasco, Nurit Engelmayer, et al.Proceedings of the National Academy of Sciences of the United States of America|March 21, 2022
Photopharmacological modulation of native CRAC channels using azoboronate photoswitchesRonald Udasin, Anwesha Sil, Elia Zomot, et al.Frontiers in Pharmacology|November 5, 2019
2-APB and CBD-Mediated Targeting of Charged Cytotoxic Compounds Into Tumor Cells Suggests the Involvement of TRPV2 ChannelsHagit Neumann-Raizel, Asaf Shilo, Shaya Lev, et al.The Journal of Clinical Investigation|December 1, 2022
Nociception and pain in humans lacking a functional TRPV1 channelBen Katz, Rachel Zaguri, Simon Edvardson, et al.Plos One|September 11, 2012
Expression of TRPV1 channels after nerve injury provides an essential delivery tool for neuropathic pain attenuationHossain Md Zakir, Rahman Md Mostafeezur, Akiko Suzuki, et al.Pageof 4