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The Journal of Biological Chemistry|August 30, 2008
Expression of S100A6 in cardiac myocytes limits apoptosis induced by tumor necrosis factor-alphaJames N Tsoporis, Shehla Izhar, Thomas G Parker
American Journal of Hypertension|August 29, 2009
S100B modulates the hemodynamic response to norepinephrine stimulationJames N Tsoporis, Christopher B Overgaard, Shehla Izhar, et al.
Journal of Molecular and Cellular Cardiology|September 6, 2011
S100B-RAGE dependent VEGF secretion by cardiac myocytes induces myofibroblast proliferationJames N Tsoporis, Shehla Izhar, Gerald Proteau, et al.
Experimental Cell Research|March 3, 2018
Deficiency of S100B confers resistance to experimental diabetes in miceForough Mohammadzadeh, James N Tsoporis, Shehla Izhar, et al.
Current Pharmaceutical Design|July 13, 2013
Conditional cardiac overexpression of S100A6 attenuates myocyte hypertrophy and apoptosis following myocardial infarctionJames T Tsoporis, Shehla Izhar, Jean-Francois Desjardins, et al.
Journal of Cardiovascular Pharmacology|May 9, 2018
Antiapoptotic Effect of β1 Blockers in Ascending Thoracic Aortic Smooth Muscle Cells: The Role of HSP70 ExpressionIoannis K Rizos, James N Tsoporis, Ioannis K Toumpoulis, et al.
International Journal of Cardiology|February 9, 2023
Emerging markers of inflammation and oxidative stress as potential predictors of coronary artery diseaseGeorge Tsirebolos, James N Tsoporis, Ioannis-Alexandros Drosatos, et al.
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