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Shenhav Cohen

Showing results (1-10 of 20) with videos related to

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Biochimica Et Biophysica Acta. Molecular Cell Research|July 1, 2020
Role of calpains in promoting desmin filaments depolymerization and muscle atrophyShenhav Cohen
Biomolecules|January 20, 2021
Breakdown of Filamentous Myofibrils by the UPS-Step by StepDina Aweida, Shenhav Cohen
EMBO Reports|October 24, 2022
The AAA-ATPase ATAD1 and its partners promote degradation of desmin intermediate filaments in muscleDina Aweida, Shenhav Cohen
The FEBS Journal|April 7, 2021
New roles for desmin in the maintenance of muscle homeostasisGiulio Agnetti, Harald Herrmann, Shenhav Cohen
Nature Reviews. Drug Discovery|January 1, 2015
Muscle wasting in disease: molecular mechanisms and promising therapiesShenhav Cohen, James A Nathan, Alfred L Goldberg
Proceedings of the National Academy of Sciences of the United States of America|January 19, 2017
Myofibril breakdown during atrophy is a delayed response requiring the transcription factor PAX4 and desmin depolymerizationAlexandra Volodin, Idit Kosti, Alfred Lewis Goldberg, et al.
The Journal of Cell Biology|August 1, 2018
GSK3-β promotes calpain-1-mediated desmin filament depolymerization and myofibril loss in atrophyDina Aweida, Inga Rudesky, Alexandra Volodin, et al.
Leukemia Research|June 28, 2005
JAK-STAT signaling involved in phorbol 12-myristate 13-acetate- and dimethyl sulfoxide-induced 2'-5' oligoadenylate synthetase expression in human HL-60 leukemia cellsShenhav Cohen, Sara Dovrat, Ronit Sarid, et al.
The Journal of Cell Biology|August 22, 2012
Ubiquitylation by Trim32 causes coupled loss of desmin, Z-bands, and thin filaments in muscle atrophyShenhav Cohen, Bo Zhai, Steven P Gygi, et al.
The Journal of Cell Biology|February 26, 2014
Trim32 reduces PI3K-Akt-FoxO signaling in muscle atrophy by promoting plakoglobin-PI3K dissociationShenhav Cohen, Donghoon Lee, Bo Zhai, et al.
Pageof 2

Showing results (1-10 of 20) with videos related to

Sort By:
Pageof 2
Biochimica Et Biophysica Acta. Molecular Cell Research|July 1, 2020
Role of calpains in promoting desmin filaments depolymerization and muscle atrophyShenhav Cohen
Biomolecules|January 20, 2021
Breakdown of Filamentous Myofibrils by the UPS-Step by StepDina Aweida, Shenhav Cohen
EMBO Reports|October 24, 2022
The AAA-ATPase ATAD1 and its partners promote degradation of desmin intermediate filaments in muscleDina Aweida, Shenhav Cohen
The FEBS Journal|April 7, 2021
New roles for desmin in the maintenance of muscle homeostasisGiulio Agnetti, Harald Herrmann, Shenhav Cohen
Nature Reviews. Drug Discovery|January 1, 2015
Muscle wasting in disease: molecular mechanisms and promising therapiesShenhav Cohen, James A Nathan, Alfred L Goldberg
Proceedings of the National Academy of Sciences of the United States of America|January 19, 2017
Myofibril breakdown during atrophy is a delayed response requiring the transcription factor PAX4 and desmin depolymerizationAlexandra Volodin, Idit Kosti, Alfred Lewis Goldberg, et al.
The Journal of Cell Biology|August 1, 2018
GSK3-β promotes calpain-1-mediated desmin filament depolymerization and myofibril loss in atrophyDina Aweida, Inga Rudesky, Alexandra Volodin, et al.
Leukemia Research|June 28, 2005
JAK-STAT signaling involved in phorbol 12-myristate 13-acetate- and dimethyl sulfoxide-induced 2'-5' oligoadenylate synthetase expression in human HL-60 leukemia cellsShenhav Cohen, Sara Dovrat, Ronit Sarid, et al.
The Journal of Cell Biology|August 22, 2012
Ubiquitylation by Trim32 causes coupled loss of desmin, Z-bands, and thin filaments in muscle atrophyShenhav Cohen, Bo Zhai, Steven P Gygi, et al.
The Journal of Cell Biology|February 26, 2014
Trim32 reduces PI3K-Akt-FoxO signaling in muscle atrophy by promoting plakoglobin-PI3K dissociationShenhav Cohen, Donghoon Lee, Bo Zhai, et al.
Pageof 2