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Shigeto Sato

Showing results (21-30 of 55) with videos related to

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FEBS Letters|February 16, 2010
PINK1 is recruited to mitochondria with parkin and associates with LC3 in mitophagySumihiro Kawajiri, Shinji Saiki, Shigeto Sato, et al.
Frontiers in Immunology|December 5, 2022
Age related immune modulation of experimental autoimmune encephalomyelitis in PINK1 knockout miceDavide Cossu, Kazumasa Yokoyama, Shigeto Sato, et al.
Scientific Reports|December 21, 2012
PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagyKahori Shiba-Fukushima, Yuzuru Imai, Shigeharu Yoshida, et al.
Biochemical and Biophysical Research Communications|April 11, 2009
Parkin stabilizes PINK1 through direct interactionKahori Shiba, Takeo Arai, Shigeto Sato, et al.
The American Journal of Pathology|October 23, 2016
Lysosomal Storage of Subunit c of Mitochondrial ATP Synthase in Brain-Specific Atp13a2-Deficient MiceShigeto Sato, Masato Koike, Manabu Funayama, et al.
FEBS Letters|January 17, 2009
Programmed cell death-2 isoform1 is ubiquitinated by parkin and increased in the substantia nigra of patients with autosomal recessive Parkinson's diseaseJiro Fukae, Shigeto Sato, Kahori Shiba, et al.
Scientific Reports|April 28, 2023
Gait improvement with wearable cyborg HAL trunk unit for parkinsonian patients: five case reportsAkira Uehara, Hiroaki Kawamoto, Hisamasa Imai, et al.
Neuroscience Letters|January 27, 2019
Loss of nuclear REST/NRSF in aged-dopaminergic neurons in Parkinson's disease patientsMiwako Kawamura, Shigeto Sato, Gen Matsumoto, et al.
Scientific Reports|February 14, 2018
Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged miceShigeto Sato, Toshiki Uchihara, Takahiro Fukuda, et al.
Neurobiology of Disease|September 7, 2010
Mitochondrial membrane potential decrease caused by loss of PINK1 is not due to proton leak, but to respiratory chain defectsTaku Amo, Shigeto Sato, Shinji Saiki, et al.
Pageof 6

Showing results (21-30 of 55) with videos related to

Sort By:
Pageof 6
FEBS Letters|February 16, 2010
PINK1 is recruited to mitochondria with parkin and associates with LC3 in mitophagySumihiro Kawajiri, Shinji Saiki, Shigeto Sato, et al.
Frontiers in Immunology|December 5, 2022
Age related immune modulation of experimental autoimmune encephalomyelitis in PINK1 knockout miceDavide Cossu, Kazumasa Yokoyama, Shigeto Sato, et al.
Scientific Reports|December 21, 2012
PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagyKahori Shiba-Fukushima, Yuzuru Imai, Shigeharu Yoshida, et al.
Biochemical and Biophysical Research Communications|April 11, 2009
Parkin stabilizes PINK1 through direct interactionKahori Shiba, Takeo Arai, Shigeto Sato, et al.
The American Journal of Pathology|October 23, 2016
Lysosomal Storage of Subunit c of Mitochondrial ATP Synthase in Brain-Specific Atp13a2-Deficient MiceShigeto Sato, Masato Koike, Manabu Funayama, et al.
FEBS Letters|January 17, 2009
Programmed cell death-2 isoform1 is ubiquitinated by parkin and increased in the substantia nigra of patients with autosomal recessive Parkinson's diseaseJiro Fukae, Shigeto Sato, Kahori Shiba, et al.
Scientific Reports|April 28, 2023
Gait improvement with wearable cyborg HAL trunk unit for parkinsonian patients: five case reportsAkira Uehara, Hiroaki Kawamoto, Hisamasa Imai, et al.
Neuroscience Letters|January 27, 2019
Loss of nuclear REST/NRSF in aged-dopaminergic neurons in Parkinson's disease patientsMiwako Kawamura, Shigeto Sato, Gen Matsumoto, et al.
Scientific Reports|February 14, 2018
Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged miceShigeto Sato, Toshiki Uchihara, Takahiro Fukuda, et al.
Neurobiology of Disease|September 7, 2010
Mitochondrial membrane potential decrease caused by loss of PINK1 is not due to proton leak, but to respiratory chain defectsTaku Amo, Shigeto Sato, Shinji Saiki, et al.
Pageof 6