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Cellular Signalling|April 7, 2022
NOX2-derived hydrogen peroxide impedes the AMPK/Akt-mTOR signaling pathway contributing to cell death in neuronal cellsRuijie Zhang, Chunxiao Liu, Liu Yang, et al.Cell Calcium|February 17, 2020
Rapamycin inhibits B-cell activating factor (BAFF)-stimulated cell proliferation and survival by suppressing Ca<sup>2+</sup>-CaMKII-dependent PTEN/Akt-Erk1/2 signaling pathway in normal and neoplastic B-lymphoid cellsQingyu Zeng, Zhihan Zhou, Shanshan Qin, et al.Neuropharmacology|September 1, 2022
Neuroprotection of resveratrol against cadmium-poisoning acts through dual inhibition of mTORC1/2 signalingChunxiao Liu, Ruijie Zhang, Liu Yang, et al.Frontiers in Oncology|May 10, 2021
Cryptotanshinone Inhibits ERα-Dependent and -Independent BCRP Oligomer Formation to Reverse Multidrug Resistance in Breast CancerWenting Ni, Hui Fan, Xiuqin Zheng, et al.Toxicological Sciences : an Official Journal of the Society of Toxicology|October 12, 2014
Rotenone induction of hydrogen peroxide inhibits mTOR-mediated S6K1 and 4E-BP1/eIF4E pathways, leading to neuronal apoptosisQian Zhou, Chunxiao Liu, Wen Liu, et al.Cells|April 27, 2026
Metformin Alleviates Cadmium-Induced Autophagic Flux Impairment-Dependent Apoptosis by Activating AMPK in Neuronal CellsWen Wu, Xiaoling Chen, Tong Ji, et al.Journal of Neurochemistry|January 28, 2017
Celastrol ameliorates Cd-induced neuronal apoptosis by targeting NOX2-derived ROS-dependent PP5-JNK signaling pathwayChong Xu, Xiaoxue Wang, Chenjian Gu, et al.Neuropharmacology|March 27, 2020
Metformin attenuates cadmium-induced neuronal apoptosis in vitro via blocking ROS-dependent PP5/AMPK-JNK signaling pathwayXiaoling Chen, Wen Wu, Baoming Gong, et al.Carcinogenesis|February 3, 2012
Curcumin inhibits protein phosphatases 2A and 5, leading to activation of mitogen-activated protein kinases and death in tumor cellsXiuzhen Han, Baoshan Xu, Christopher S Beevers, et al.British Journal of Cancer|September 6, 2017
SKLB188 inhibits the growth of head and neck squamous cell carcinoma by suppressing EGFR signallingMansoureh Barzegar, Shuang Ma, Chao Zhang, et al.Pageof 15