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The Journal of Biological Chemistry|June 21, 2019
Interferon-γ induces the cell surface exposure of phosphatidylserine by activating the protein MLKL in the absence of caspase-8 activityJiancheng Chen, Shunsuke Kuroki, Masataka Someda, et al.Genes to Cells : Devoted to Molecular & Cellular Mechanisms|January 27, 2006
Human T-cell leukemia virus type-I oncoprotein Tax inhibits Fas-mediated apoptosis by inducing cellular FLIP through activation of NF-kappaBKazuo Okamoto, Jun-ichi Fujisawa, Michael Reth, et al.Molecular and Cellular Biology|October 18, 2005
Viral FLIP enhances Wnt signaling downstream of stabilized beta-catenin, leading to control of cell growthShiho Nakagiri, Akira Murakami, Shinji Takada, et al.Molecular and Cellular Biology|June 24, 2009
Interaction of FLASH with arsenite resistance protein 2 is involved in cell cycle progression at S phaseMaria Kiriyama, Yohei Kobayashi, Motoki Saito, et al.The Journal of Biological Chemistry|May 26, 2004
A caspase-8-independent signaling pathway activated by Fas ligation leads to exposure of the Bak N terminusLilin Zhang, Shigeomi Shimizu, Kazuhiro Sakamaki, et al.Cell Death and Differentiation|October 30, 2019
Caspase-8, receptor-interacting protein kinase 1 (RIPK1), and RIPK3 regulate retinoic acid-induced cell differentiation and necroptosisMasataka Someda, Shunsuke Kuroki, Hitoshi Miyachi, et al.The Journal of Biological Chemistry|November 6, 2002
Translocation of analogues of the antimicrobial peptides magainin and buforin across human cell membranesKenta Takeshima, Akinori Chikushi, Kyung-Kwon Lee, et al.Current Biology : CB|June 20, 2006
Continuous ERK activation downregulates antiproliferative genes throughout G1 phase to allow cell-cycle progressionTakuya Yamamoto, Miki Ebisuya, Fumito Ashida, et al.Japanese Journal of Cancer Research : Gann|November 6, 2002
Phosphorylation of Fas-associated death domain contributes to enhancement of etoposide-induced apoptosis in prostate cancer cellsKeiji Shimada, Mitsutoshi Nakamura, Eiwa Ishida, et al.Arthritis and Rheumatism|October 4, 2005
Pathogenic role of the CXCL16-CXCR6 pathway in rheumatoid arthritisToshihiro Nanki, Takeshi Shimaoka, Kenji Hayashida, et al.Pageof 6