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Molecular and Cellular Biology|January 6, 2017
<i>KLHL3</i> Knockout Mice Reveal the Physiological Role of KLHL3 and the Pathophysiology of Pseudohypoaldosteronism Type II Caused by Mutant KLHL3Emi Sasaki, Koichiro Susa, Takayasu Mori, et al.
Aging|October 27, 2023
Chronic kidney disease causes blood-brain barrier breakdown via urea-activated matrix metalloproteinase-2 and insolubility of tau proteinHisazumi Matsuki, Shintaro Mandai, Hiroki Shiwaku, et al.
Clinical and Experimental Nephrology|February 17, 2021
Deletion of Alox15 improves kidney dysfunction and inhibits fibrosis by increased PGD<sub>2</sub> in the kidneyNaohiro Takahashi, Hiroaki Kikuchi, Ayaka Usui, et al.
International Journal of Rheumatic Diseases|March 7, 2023
Thrombocytopenia during avacopan administration: A case reportNobuhisa Morimoto, Takayasu Mori, Shingo Shioji, et al.
Kidney International|February 16, 2020
Renal TNFα activates the WNK phosphorylation cascade and contributes to salt-sensitive hypertension in chronic kidney diseaseTaisuke Furusho, Eisei Sohara, Shintaro Mandai, et al.
Scientific Reports|March 2, 2024
Impact of COVID-19 versus other pneumonia on in-hospital mortality and functional decline among Japanese dialysis patients: a retrospective cohort studyKen Ikenouchi, Daiei Takahashi, Shintaro Mandai, et al.
Kidney International|November 21, 2018
Failure to sense energy depletion may be a novel therapeutic target in chronic kidney diseaseHiroaki Kikuchi, Emi Sasaki, Naohiro Nomura, et al.
Circulation Research|January 26, 2023
Circulating Extracellular Vesicle-Propagated microRNA Signature as a Vascular Calcification Factor in Chronic Kidney DiseaseTakaaki Koide, Shintaro Mandai, Reo Kitaoka, et al.
Journal of the American Heart Association|December 10, 2025
Circulating Extracellular Vesicle MicroRNAs as Predictive Biomarkers for Kidney and Cardiovascular EventsShunsuke Inaba, Takanori Hasegawa, Yuta Nakano, et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|November 1, 2022
Absence of ULK1 decreases AMPK activity in the kidney, leading to chronic kidney disease progressionTomoki Yanagi, Hiroaki Kikuchi, Koichiro Susa, et al.
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