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Journal of Molecular Biology|June 7, 2011
EPR spectra and molecular dynamics agree that the nucleotide pocket of myosin V is closed and that it opens on binding actinThomas J Purcell, Nariman Naber, Shirley Sutton, et al.
Science Advances|March 2, 2017
Biophysical properties of human β-cardiac myosin with converter mutations that cause hypertrophic cardiomyopathyMasataka Kawana, Saswata S Sarkar, Shirley Sutton, et al.
Cell Reports|May 5, 2015
Ensemble force changes that result from human cardiac myosin mutations and a small-molecule effectorTural Aksel, Elizabeth Choe Yu, Shirley Sutton, et al.
Plos One|December 25, 2013
Effects of troponin T cardiomyopathy mutations on the calcium sensitivity of the regulated thin filament and the actomyosin cross-bridge kinetics of human β-cardiac myosinRuth F Sommese, Suman Nag, Shirley Sutton, et al.
Plos Biology|October 20, 2004
Dictyostelium myosin bipolar thick filament formation: importance of charge and specific domains of the myosin rodDaniel Hostetter, Sarah Rice, Sara Dean, et al.
Nature Communications|August 5, 2015
Harmonic force spectroscopy measures load-dependent kinetics of individual human β-cardiac myosin moleculesJongmin Sung, Suman Nag, Kim I Mortensen, et al.
Nature Structural & Molecular Biology|May 9, 2017
The myosin mesa and the basis of hypercontractility caused by hypertrophic cardiomyopathy mutationsSuman Nag, Darshan V Trivedi, Saswata S Sarkar, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 27, 2013
Molecular consequences of the R453C hypertrophic cardiomyopathy mutation on human β-cardiac myosin motor functionRuth F Sommese, Jongmin Sung, Suman Nag, et al.
BMC Medical Genetics|October 27, 2015
Establishing disease causality for a novel gene variant in familial dilated cardiomyopathy using a functional in-vitro assay of regulated thin filaments and human cardiac myosinStephen Pan, Ruth F Sommese, Karim I Sallam, et al.
Science Advances|November 25, 2015
Contractility parameters of human β-cardiac myosin with the hypertrophic cardiomyopathy mutation R403Q show loss of motor functionSuman Nag, Ruth F Sommese, Zoltan Ujfalusi, et al.
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