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Shizu Hayashi

Showing results (11-20 of 27) with videos related to

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American Journal of Physiology. Lung Cellular and Molecular Physiology|March 9, 2004
Contribution of IL-1 beta and TNF-alpha to the initiation of the peripheral lung response to atmospheric particulates (PM10)Hiroshi Ishii, Takeshi Fujii, James C Hogg, et al.
COPD|December 2, 2006
A lung tissue bank for gene expression studies in chronic obstructive pulmonary diseaseLily Ding, Kevin B Quinlan, W Mark Elliott, et al.
American Journal of Respiratory and Critical Care Medicine|July 18, 2002
Inflammatory mediator mRNA expression by adenovirus E1A-transfected bronchial epithelial cellsYuji Higashimoto, W Mark Elliott, Ali R Behzad, et al.
Toxicology and Applied Pharmacology|August 28, 2007
Calcium dependent and independent cytokine synthesis by air pollution particle-exposed human bronchial epithelial cellsNoriho Sakamoto, Shizu Hayashi, John Gosselink, et al.
American Journal of Respiratory Cell and Molecular Biology|July 2, 2002
Interaction of alveolar macrophages and airway epithelial cells following exposure to particulate matter produces mediators that stimulate the bone marrowTakeshi Fujii, Shizu Hayashi, James C Hogg, et al.
American Journal of Respiratory Cell and Molecular Biology|December 26, 2001
Emphysematous lung destruction by cigarette smoke. The effects of latent adenoviral infection on the lung inflammatory responseBernard Meshi, Timothy Z Vitalis, Diana Ionescu, et al.
Chest|June 11, 2011
Patterns of retention of particulate matter in lung tissues of patients with COPD: potential role in disease progressionSean H Ling, John E McDonough, John V Gosselink, et al.
Respiratory Research|August 2, 2005
Alveolar macrophage-epithelial cell interaction following exposure to atmospheric particles induces the release of mediators involved in monocyte mobilization and recruitmentHiroshi Ishii, Shizu Hayashi, James C Hogg, et al.
Respiratory Medicine|June 9, 2009
Role of genetic susceptibility to latent adenoviral infection and decreased lung functionIkuma Kasuga, James C Hogg, Peter D Paré, et al.
The New England Journal of Medicine|May 13, 2005
Decreased histone deacetylase activity in chronic obstructive pulmonary diseaseKazuhiro Ito, Misako Ito, W Mark Elliott, et al.
Pageof 3

Showing results (11-20 of 27) with videos related to

Sort By:
Pageof 3
American Journal of Physiology. Lung Cellular and Molecular Physiology|March 9, 2004
Contribution of IL-1 beta and TNF-alpha to the initiation of the peripheral lung response to atmospheric particulates (PM10)Hiroshi Ishii, Takeshi Fujii, James C Hogg, et al.
COPD|December 2, 2006
A lung tissue bank for gene expression studies in chronic obstructive pulmonary diseaseLily Ding, Kevin B Quinlan, W Mark Elliott, et al.
American Journal of Respiratory and Critical Care Medicine|July 18, 2002
Inflammatory mediator mRNA expression by adenovirus E1A-transfected bronchial epithelial cellsYuji Higashimoto, W Mark Elliott, Ali R Behzad, et al.
Toxicology and Applied Pharmacology|August 28, 2007
Calcium dependent and independent cytokine synthesis by air pollution particle-exposed human bronchial epithelial cellsNoriho Sakamoto, Shizu Hayashi, John Gosselink, et al.
American Journal of Respiratory Cell and Molecular Biology|July 2, 2002
Interaction of alveolar macrophages and airway epithelial cells following exposure to particulate matter produces mediators that stimulate the bone marrowTakeshi Fujii, Shizu Hayashi, James C Hogg, et al.
American Journal of Respiratory Cell and Molecular Biology|December 26, 2001
Emphysematous lung destruction by cigarette smoke. The effects of latent adenoviral infection on the lung inflammatory responseBernard Meshi, Timothy Z Vitalis, Diana Ionescu, et al.
Chest|June 11, 2011
Patterns of retention of particulate matter in lung tissues of patients with COPD: potential role in disease progressionSean H Ling, John E McDonough, John V Gosselink, et al.
Respiratory Research|August 2, 2005
Alveolar macrophage-epithelial cell interaction following exposure to atmospheric particles induces the release of mediators involved in monocyte mobilization and recruitmentHiroshi Ishii, Shizu Hayashi, James C Hogg, et al.
Respiratory Medicine|June 9, 2009
Role of genetic susceptibility to latent adenoviral infection and decreased lung functionIkuma Kasuga, James C Hogg, Peter D Paré, et al.
The New England Journal of Medicine|May 13, 2005
Decreased histone deacetylase activity in chronic obstructive pulmonary diseaseKazuhiro Ito, Misako Ito, W Mark Elliott, et al.
Pageof 3