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Shohreh Varmeh

Showing results (1-10 of 20) with videos related to

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The Journal of Biological Chemistry|June 27, 2007
The dual specificity phosphatase Cdc25B, but not the closely related Cdc25C, is capable of inhibiting cellular proliferation in a manner dependent upon its catalytic activityShohreh Varmeh-Ziaie, James J Manfredi
The Journal of Biological Chemistry|January 13, 2009
Inappropriate activation of cyclin-dependent kinases by the phosphatase Cdc25b results in premature mitotic entry and triggers a p53-dependent checkpointShohreh Varmeh, James J Manfredi
Molecular Cancer Therapeutics|December 17, 2008
Overexpression of the dual specificity phosphatase, Cdc25C, confers sensitivity on tumor cells to doxorubicin-induced cell deathShohreh Varmeh, James J Manfredi
Genes & Cancer|June 28, 2012
Cellular senescence as a possible mechanism for halting progression of keloid lesionsShohreh Varmeh, Ainara Egia, Duncan McGrouther, et al.
The American Journal of Pathology|September 18, 2012
Keloid disease can be inhibited by antagonizing excessive mTOR signaling with a novel dual TORC1/2 inhibitorFarhatullah Syed, David Sherris, Ralf Paus, et al.
Oncotarget|July 5, 2014
Combined BRAF(V600E)- and SRC-inhibition induces apoptosis, evokes an immune response and reduces tumor growth in an immunocompetent orthotopic mouse model of anaplastic thyroid cancerPierre Vanden Borre, Viswanath Gunda, David G McFadden, et al.
Surgery|October 13, 2015
Genome-wide analysis of differentially expressed miRNA in PLX4720-resistant and parental human thyroid cancer cell linesShohreh Varmeh, Pierre Vanden Borre, Viswanath Gunda, et al.
Thyroid : Official Journal of the American Thyroid Association|December 4, 2013
The next generation of orthotopic thyroid cancer models: immunocompetent orthotopic mouse models of BRAF V600E-positive papillary and anaplastic thyroid carcinomaPierre Vanden Borre, David G McFadden, Viswanath Gunda, et al.
Molecular Cell|December 3, 2004
DNA damage-induced downregulation of Cdc25C is mediated by p53 via two independent mechanisms: one involves direct binding to the cdc25C promoterSelvon St Clair, Luciana Giono, Shohreh Varmeh-Ziaie, et al.
Nature Genetics|August 22, 2020
Author Correction: Aberrant ERG expression cooperates with loss of PTEN to promote cancer progression in the prostateBrett S Carver, Jennifer Tran, Anuradha Gopalan, et al.
Pageof 2

Showing results (1-10 of 20) with videos related to

Sort By:
Pageof 2
The Journal of Biological Chemistry|June 27, 2007
The dual specificity phosphatase Cdc25B, but not the closely related Cdc25C, is capable of inhibiting cellular proliferation in a manner dependent upon its catalytic activityShohreh Varmeh-Ziaie, James J Manfredi
The Journal of Biological Chemistry|January 13, 2009
Inappropriate activation of cyclin-dependent kinases by the phosphatase Cdc25b results in premature mitotic entry and triggers a p53-dependent checkpointShohreh Varmeh, James J Manfredi
Molecular Cancer Therapeutics|December 17, 2008
Overexpression of the dual specificity phosphatase, Cdc25C, confers sensitivity on tumor cells to doxorubicin-induced cell deathShohreh Varmeh, James J Manfredi
Genes & Cancer|June 28, 2012
Cellular senescence as a possible mechanism for halting progression of keloid lesionsShohreh Varmeh, Ainara Egia, Duncan McGrouther, et al.
The American Journal of Pathology|September 18, 2012
Keloid disease can be inhibited by antagonizing excessive mTOR signaling with a novel dual TORC1/2 inhibitorFarhatullah Syed, David Sherris, Ralf Paus, et al.
Oncotarget|July 5, 2014
Combined BRAF(V600E)- and SRC-inhibition induces apoptosis, evokes an immune response and reduces tumor growth in an immunocompetent orthotopic mouse model of anaplastic thyroid cancerPierre Vanden Borre, Viswanath Gunda, David G McFadden, et al.
Surgery|October 13, 2015
Genome-wide analysis of differentially expressed miRNA in PLX4720-resistant and parental human thyroid cancer cell linesShohreh Varmeh, Pierre Vanden Borre, Viswanath Gunda, et al.
Thyroid : Official Journal of the American Thyroid Association|December 4, 2013
The next generation of orthotopic thyroid cancer models: immunocompetent orthotopic mouse models of BRAF V600E-positive papillary and anaplastic thyroid carcinomaPierre Vanden Borre, David G McFadden, Viswanath Gunda, et al.
Molecular Cell|December 3, 2004
DNA damage-induced downregulation of Cdc25C is mediated by p53 via two independent mechanisms: one involves direct binding to the cdc25C promoterSelvon St Clair, Luciana Giono, Shohreh Varmeh-Ziaie, et al.
Nature Genetics|August 22, 2020
Author Correction: Aberrant ERG expression cooperates with loss of PTEN to promote cancer progression in the prostateBrett S Carver, Jennifer Tran, Anuradha Gopalan, et al.
Pageof 2