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Archives of Toxicology|June 8, 2018
Autophagy and acetaminophen-induced hepatotoxicityShulin Shan, Zhenyu Shen, Fuyong Song
Cerebral Cortex (New York, N.Y. : 1991)|January 8, 2024
Carbon disulfide induces accumulation of TDP-43 in the cytoplasm and mitochondrial dysfunction in rat spinal cordsZhidan Liu, Yalong Qiang, Shulin Shan, et al.
Toxicology Research|November 7, 2022
MitoQ alleviates carbon tetrachloride-induced liver fibrosis in mice through regulating JNK/YAP pathwayShulin Shan, Zhaoxiong Liu, Zhidan Liu, et al.
Human & Experimental Toxicology|June 17, 2022
Calpain-mediated cleavage of mitochondrial fusion/fission proteins in acetaminophen-induced mice liver injuryShulin Shan, Zhaoxiong Liu, Linlin Li, et al.
Neurotoxicity Research|March 2, 2021
Atg7 Knockout Alleviated the Axonal Injury of Neuro-2a Cells Induced by Tri-Ortho-Cresyl PhosphateCuiqin Zhang, Kang Kang, Yisi Chen, et al.
Biochemical Pharmacology|September 23, 2019
Mitophagy protects against acetaminophen-induced acute liver injury in mice through inhibiting NLRP3 inflammasome activationShulin Shan, Zhenyu Shen, Cuiqin Zhang, et al.
Toxicology Research|July 5, 2022
Drp1-mediated mitochondrial fission promotes carbon tetrachloride-induced hepatic fibrogenesis in miceShulin Shan, Zhidan Liu, Shuai Wang, et al.
Ecotoxicology and Environmental Safety|June 22, 2024
Mitochondrial transfer of α-synuclein mediates carbon disulfide-induced mitochondrial dysfunction and neurotoxicityZhidan Liu, Shulin Shan, Kang Kang, et al.
Ecotoxicology and Environmental Safety|August 30, 2023
Mitochondrial oxidative stress regulates LonP1-TDP-43 pathway and rises mitochondrial damage in carbon tetrachloride-induced liver fibrosisShulin Shan, Zhidan Liu, Shuai Wang, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|November 23, 2022
Mitochondrial dysfunction promotes the necroptosis of Purkinje cells in the cerebellum of acrylamide-exposed ratsZhengcheng Huang, Shuai Wang, Yiyu Yang, et al.
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