Search research articles
Contact Us
Filters
Showing results (21-30 of 40) with videos related to
Page
of 4
Sort By:
Oncotarget
|
September 5, 2015
Dissecting the contribution of EBNA3C domains important for EBV-induced B-cell growth and proliferation
Hem Chandra Jha, Sanket Kumar Shukla, Jie Lu, et al.
Scientific Reports
|
September 14, 2016
Crocetin exploits p53-induced death domain (PIDD) and FAS-associated death domain (FADD) proteins to induce apoptosis in colorectal cancer
Pallab Ray, Deblina Guha, Juni Chakraborty, et al.
Plos Pathogens
|
November 9, 2013
IRF-4-mediated CIITA transcription is blocked by KSHV encoded LANA to inhibit MHC II presentation
Qiliang Cai, Shuvomoy Banerjee, Amanda Cervini, et al.
Journal of Virology
|
January 17, 2014
Epstein-Barr virus essential antigen EBNA3C attenuates H2AX expression
Hem C Jha, Mahadesh Prasad A J, Abhik Saha, et al.
Plos Pathogens
|
May 10, 2013
The EBV Latent Antigen 3C Inhibits Apoptosis through Targeted Regulation of Interferon Regulatory Factors 4 and 8
Shuvomoy Banerjee, Jie Lu, Qiliang Cai, et al.
Plos Pathogens
|
December 17, 2011
Epstein-Barr virus nuclear antigen 3C stabilizes Gemin3 to block p53-mediated apoptosis
Qiliang Cai, Yi Guo, Bingyi Xiao, et al.
Pathology, Research and Practice
|
August 5, 2023
Virus-associated neuroendocrine cancers: Pathogenesis and current therapeutics
Juni Banerjee, Ramya P Ranjan, Md Tanjim Alam, et al.
Journal of Virology
|
January 31, 2014
Kaposi's sarcoma-associated herpesvirus-encoded LANA contributes to viral latent replication by activating phosphorylation of survivin
Jie Lu, Hem C Jha, Subhash C Verma, et al.
Journal of Virology
|
August 30, 2013
EBNA3C-mediated regulation of aurora kinase B contributes to Epstein-Barr virus-induced B-cell proliferation through modulation of the activities of the retinoblastoma protein and apoptotic caspases
Hem Chandra Jha, Jie Lu, Abhik Saha, et al.
The Journal of Biological Chemistry
|
August 3, 2010
Gain of cellular adaptation due to prolonged p53 impairment leads to functional switchover from p53 to p73 during DNA damage in acute myeloid leukemia cells
Juni Chakraborty, Shuvomoy Banerjee, Pallab Ray, et al.
Page
of 4
Search research articles
Search
Showing results (21-30 of 40) with videos related to
Sort By:
Page
of 4
Oncotarget
|
September 5, 2015
Dissecting the contribution of EBNA3C domains important for EBV-induced B-cell growth and proliferation
Hem Chandra Jha, Sanket Kumar Shukla, Jie Lu, et al.
Scientific Reports
|
September 14, 2016
Crocetin exploits p53-induced death domain (PIDD) and FAS-associated death domain (FADD) proteins to induce apoptosis in colorectal cancer
Pallab Ray, Deblina Guha, Juni Chakraborty, et al.
Plos Pathogens
|
November 9, 2013
IRF-4-mediated CIITA transcription is blocked by KSHV encoded LANA to inhibit MHC II presentation
Qiliang Cai, Shuvomoy Banerjee, Amanda Cervini, et al.
Journal of Virology
|
January 17, 2014
Epstein-Barr virus essential antigen EBNA3C attenuates H2AX expression
Hem C Jha, Mahadesh Prasad A J, Abhik Saha, et al.
Plos Pathogens
|
May 10, 2013
The EBV Latent Antigen 3C Inhibits Apoptosis through Targeted Regulation of Interferon Regulatory Factors 4 and 8
Shuvomoy Banerjee, Jie Lu, Qiliang Cai, et al.
Plos Pathogens
|
December 17, 2011
Epstein-Barr virus nuclear antigen 3C stabilizes Gemin3 to block p53-mediated apoptosis
Qiliang Cai, Yi Guo, Bingyi Xiao, et al.
Pathology, Research and Practice
|
August 5, 2023
Virus-associated neuroendocrine cancers: Pathogenesis and current therapeutics
Juni Banerjee, Ramya P Ranjan, Md Tanjim Alam, et al.
Journal of Virology
|
January 31, 2014
Kaposi's sarcoma-associated herpesvirus-encoded LANA contributes to viral latent replication by activating phosphorylation of survivin
Jie Lu, Hem C Jha, Subhash C Verma, et al.
Journal of Virology
|
August 30, 2013
EBNA3C-mediated regulation of aurora kinase B contributes to Epstein-Barr virus-induced B-cell proliferation through modulation of the activities of the retinoblastoma protein and apoptotic caspases
Hem Chandra Jha, Jie Lu, Abhik Saha, et al.
The Journal of Biological Chemistry
|
August 3, 2010
Gain of cellular adaptation due to prolonged p53 impairment leads to functional switchover from p53 to p73 during DNA damage in acute myeloid leukemia cells
Juni Chakraborty, Shuvomoy Banerjee, Pallab Ray, et al.
Page
of 4