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Journal of the Formosan Medical Association = Taiwan Yi Zhi|December 4, 2010
Possible mechanism of betel-quid-extract-induced expression of matrix metalloproteinase-2Yu-Chi Liu, Mei-Huei Lin, Shyun-Yeu Liu, et al.
Head & Neck|January 30, 2018
Risk analysis of malignant potential of oral verrucous hyperplasia: A follow-up study of 269 patients and copy number variation analysisMing-Heng Wu, Ji-Dung Luo, Wen-Chang Wang, et al.
American Journal of Cancer Research|October 18, 2021
Obesity-associated leptin promotes chemoresistance in colorectal cancer through YAP-dependent AXL upregulationYing-Chen Chen, Chu-Yen Chien, Chia-Chen Hsu, et al.
Molecular Medicine Reports|October 22, 2014
Sprouty2 protein is downregulated in human squamous cell carcinoma of the head and neck and suppresses cell proliferation in vitroChiang-Liang Lin, Wei-Fan Chiang, Chao-Ling Tung, et al.
Oral Oncology|February 14, 2009
Serine protease inhibitor (SERPIN) B1 promotes oral cancer cell motility and is over-expressed in invasive oral squamous cell carcinomaMei-Yu Tseng, Shyun-Yeu Liu, Hau-Ren Chen, et al.
Journal of the Formosan Medical Association = Taiwan Yi Zhi|July 23, 2005
Increased expression of matrix metalloproteinase-2 in oral cells after short-term stimulation and long-term usage of areca quidShyun-Yeu Liu, Mei-Huei Lin, Shun-Chun Yang, et al.
Neoplasia (New York, N.Y.)|December 3, 2008
Src family kinases mediate betel quid-induced oral cancer cell motility and could be a biomarker for early invasion in oral squamous cell carcinomaJeff Yi-Fu Chen, Chih-Chang Hung, Kai-Lieh Huang, et al.
Cancer Cell International|September 1, 2020
Correction to: Gamma synuclein is a novel nicotine responsive protein in oral cancer malignancyChia-Chen Hsu, Yu-Fu Su, Kuo-Yang Tsai, et al.
Cancer Cell International|July 17, 2020
Gamma synuclein is a novel nicotine responsive protein in oral cancer malignancyChia-Chen Hsu, Yu-Fu Su, Kuo-Yang Tsai, et al.
Oral Oncology|October 6, 2010
Autophagy induction by the 30-100kDa fraction of areca nut in both normal and malignant cells through reactive oxygen speciesMei-Huei Lin, Wan-Fang Hsieh, Wei-Fan Chiang, et al.
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