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Sigmar Stricker

Showing results (41-50 of 79) with videos related to

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Genes to Cells : Devoted to Molecular & Cellular Mechanisms|December 1, 2004
Modulation of GDF5/BRI-b signalling through interaction with the tyrosine kinase receptor Ror2Marei Sammar, Sigmar Stricker, Georg C Schwabe, et al.
Plos Genetics|March 29, 2008
Evolution of a core gene network for skeletogenesis in chordatesJochen Hecht, Sigmar Stricker, Ulrike Wiecha, et al.
Human Molecular Genetics|April 12, 2011
Neurofibromin (Nf1) is required for skeletal muscle developmentNadine Kossler, Sigmar Stricker, Christian Rödelsperger, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 3, 2003
Mutations in bone morphogenetic protein receptor 1B cause brachydactyly type A2Katarina Lehmann, Petra Seemann, Sigmar Stricker, et al.
Nature|March 3, 2009
A mutation in Ihh that causes digit abnormalities alters its signalling capacity and rangeBo Gao, Jianxin Hu, Sigmar Stricker, et al.
Human Molecular Genetics|February 24, 2007
Multiple roles for neurofibromin in skeletal development and growthMateusz Kolanczyk, Nadine Kossler, Jirko Kühnisch, et al.
Development (Cambridge, England)|June 5, 2023
Inhibitory SMAD6 interferes with BMP-dependent generation of muscle progenitor cells and perturbs proximodistal pattern of murine limb musclesHasan Asfour, Estelle Hirsinger, Raquel Rouco, et al.
The Journal of Clinical Investigation|April 21, 2005
An inversion involving the mouse Shh locus results in brachydactyly through dysregulation of Shh expressionMichael Niedermaier, Georg C Schwabe, Stephan Fees, et al.
The Journal of Clinical Investigation|August 30, 2005
Activating and deactivating mutations in the receptor interaction site of GDF5 cause symphalangism or brachydactyly type A2Petra Seemann, Raphaela Schwappacher, Klaus W Kjaer, et al.
Nature Communications|January 7, 2026
The dorsal aortic compartment is a developmental source of brown adipose tissue in miceSophie Heider, Cornelius Fischer, Ali Kerim Secener, et al.
Pageof 8

Showing results (41-50 of 79) with videos related to

Sort By:
Pageof 8
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|December 1, 2004
Modulation of GDF5/BRI-b signalling through interaction with the tyrosine kinase receptor Ror2Marei Sammar, Sigmar Stricker, Georg C Schwabe, et al.
Plos Genetics|March 29, 2008
Evolution of a core gene network for skeletogenesis in chordatesJochen Hecht, Sigmar Stricker, Ulrike Wiecha, et al.
Human Molecular Genetics|April 12, 2011
Neurofibromin (Nf1) is required for skeletal muscle developmentNadine Kossler, Sigmar Stricker, Christian Rödelsperger, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 3, 2003
Mutations in bone morphogenetic protein receptor 1B cause brachydactyly type A2Katarina Lehmann, Petra Seemann, Sigmar Stricker, et al.
Nature|March 3, 2009
A mutation in Ihh that causes digit abnormalities alters its signalling capacity and rangeBo Gao, Jianxin Hu, Sigmar Stricker, et al.
Human Molecular Genetics|February 24, 2007
Multiple roles for neurofibromin in skeletal development and growthMateusz Kolanczyk, Nadine Kossler, Jirko Kühnisch, et al.
Development (Cambridge, England)|June 5, 2023
Inhibitory SMAD6 interferes with BMP-dependent generation of muscle progenitor cells and perturbs proximodistal pattern of murine limb musclesHasan Asfour, Estelle Hirsinger, Raquel Rouco, et al.
The Journal of Clinical Investigation|April 21, 2005
An inversion involving the mouse Shh locus results in brachydactyly through dysregulation of Shh expressionMichael Niedermaier, Georg C Schwabe, Stephan Fees, et al.
The Journal of Clinical Investigation|August 30, 2005
Activating and deactivating mutations in the receptor interaction site of GDF5 cause symphalangism or brachydactyly type A2Petra Seemann, Raphaela Schwappacher, Klaus W Kjaer, et al.
Nature Communications|January 7, 2026
The dorsal aortic compartment is a developmental source of brown adipose tissue in miceSophie Heider, Cornelius Fischer, Ali Kerim Secener, et al.
Pageof 8