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Sigrun Roeber

Showing results (51-60 of 80) with videos related to

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European Journal of Nuclear Medicine and Molecular Imaging|March 18, 2025
Exploring the origins of frequent tau-PET signal in vermal and adjacent regionsAgnes Kling, Julia Kusche-Palenga, Carla Palleis, et al.
American Journal of Human Genetics|October 11, 2011
Absence of an orphan mitochondrial protein, c19orf12, causes a distinct clinical subtype of neurodegeneration with brain iron accumulationMonika B Hartig, Arcangela Iuso, Tobias Haack, et al.
Journal of Neuroinflammation|June 10, 2026
Ischemic injury triggers a protective microglial phenotype in models of Aβ pathologyMichael Candlish, Jan Hofmann, Desirée Brösamle, et al.
Acta Neuropathologica|May 9, 2020
Distribution patterns of tau pathology in progressive supranuclear palsyGabor G Kovacs, Milica Jecmenica Lukic, David J Irwin, et al.
Neurology|July 31, 2012
MRI characteristics and scoring in HDLS due to CSF1R gene mutationsChristina Sundal, Jay A Van Gerpen, Alexandra M Nicholson, et al.
Parkinsonism & Related Disorders|June 22, 2013
Parkinsonian features in hereditary diffuse leukoencephalopathy with spheroids (HDLS) and CSF1R mutationsChristina Sundal, Shinsuke Fujioka, Jay A Van Gerpen, et al.
Nature Medicine|February 6, 2019
Publisher Correction: ApoE attenuates unresolvable inflammation by complex formation with activated C1qChangjun Yin, Susanne Ackermann, Zhe Ma, et al.
Neuron|February 1, 2024
APOE loss-of-function variants: Compatible with longevity and associated with resistance to Alzheimer's disease pathologyAugustine Chemparathy, Yann Le Guen, Sunny Chen, et al.
Nature Medicine|January 30, 2019
ApoE attenuates unresolvable inflammation by complex formation with activated C1qChangjun Yin, Susanne Ackermann, Zhe Ma, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|November 17, 2017
Chitotriosidase (CHIT1) is increased in microglia and macrophages in spinal cord of amyotrophic lateral sclerosis and cerebrospinal fluid levels correlate with disease severity and progressionPetra Steinacker, Federico Verde, Lubin Fang, et al.
Pageof 8

Showing results (51-60 of 80) with videos related to

Sort By:
Pageof 8
European Journal of Nuclear Medicine and Molecular Imaging|March 18, 2025
Exploring the origins of frequent tau-PET signal in vermal and adjacent regionsAgnes Kling, Julia Kusche-Palenga, Carla Palleis, et al.
American Journal of Human Genetics|October 11, 2011
Absence of an orphan mitochondrial protein, c19orf12, causes a distinct clinical subtype of neurodegeneration with brain iron accumulationMonika B Hartig, Arcangela Iuso, Tobias Haack, et al.
Journal of Neuroinflammation|June 10, 2026
Ischemic injury triggers a protective microglial phenotype in models of Aβ pathologyMichael Candlish, Jan Hofmann, Desirée Brösamle, et al.
Acta Neuropathologica|May 9, 2020
Distribution patterns of tau pathology in progressive supranuclear palsyGabor G Kovacs, Milica Jecmenica Lukic, David J Irwin, et al.
Neurology|July 31, 2012
MRI characteristics and scoring in HDLS due to CSF1R gene mutationsChristina Sundal, Jay A Van Gerpen, Alexandra M Nicholson, et al.
Parkinsonism & Related Disorders|June 22, 2013
Parkinsonian features in hereditary diffuse leukoencephalopathy with spheroids (HDLS) and CSF1R mutationsChristina Sundal, Shinsuke Fujioka, Jay A Van Gerpen, et al.
Nature Medicine|February 6, 2019
Publisher Correction: ApoE attenuates unresolvable inflammation by complex formation with activated C1qChangjun Yin, Susanne Ackermann, Zhe Ma, et al.
Neuron|February 1, 2024
APOE loss-of-function variants: Compatible with longevity and associated with resistance to Alzheimer's disease pathologyAugustine Chemparathy, Yann Le Guen, Sunny Chen, et al.
Nature Medicine|January 30, 2019
ApoE attenuates unresolvable inflammation by complex formation with activated C1qChangjun Yin, Susanne Ackermann, Zhe Ma, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|November 17, 2017
Chitotriosidase (CHIT1) is increased in microglia and macrophages in spinal cord of amyotrophic lateral sclerosis and cerebrospinal fluid levels correlate with disease severity and progressionPetra Steinacker, Federico Verde, Lubin Fang, et al.
Pageof 8