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Physiology (Bethesda, Md.)|July 27, 2006
Mitochondrial uncoupling: a key contributor to reduced cardiac efficiency in diabetesSihem Boudina, E Dale AbelCirculation|October 26, 2005
Reduced mitochondrial oxidative capacity and increased mitochondrial uncoupling impair myocardial energetics in obesitySihem Boudina, Sandra Sena, Brian T O'Neill, et al.Current Pharmaceutical Design|March 2, 2013
Cardiac aging and insulin resistance: could insulin/insulin-like growth factor (IGF) signaling be used as a therapeutic target?Sihem BoudinaCurrent Pharmaceutical Design|January 18, 2013
Cardiac dysfunction and oxidative stress in the metabolic syndrome: an update on antioxidant therapiesOlesya Ilkun, Sihem BoudinaJournal of Molecular and Cellular Cardiology|October 9, 2017
Activation of IGF-1 receptors and Akt signaling by systemic hyperinsulinemia contributes to cardiac hypertrophy but does not regulate cardiac autophagy in obese diabetic miceKarla Maria Pires, Marcio Buffolo, Christin Schaaf, et al.The FEBS Journal|January 7, 2012
Cytosolic, but not mitochondrial, oxidative stress is a likely contributor to cardiac hypertrophy resulting from cardiac specific GLUT4 deletion in miceYan Li, Adam R Wende, Orathai Nunthakungwan, et al.Diabetes|August 23, 2012
UCP3 regulates cardiac efficiency and mitochondrial coupling in high fat-fed mice but not in leptin-deficient miceSihem Boudina, Yong Hwan Han, Shaobo Pei, et al.Frontiers in Physiology|November 19, 2021
The Effects of Exercise on White and Brown Adipose Tissue Cellularity, Metabolic Activity and RemodelingJacob D Garritson, Sihem BoudinaReviews in Endocrine & Metabolic Disorders|February 25, 2010
Diabetic cardiomyopathy, causes and effectsSihem Boudina, Evan Dale AbelPageof 32