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Molecular Oncology|September 16, 2025
Inhibition of CDK9 enhances AML cell death induced by combined venetoclax and azacitidineShuangshuang Wu, Jianlei Zhao, Aaban Asfar Azmi, et al.Journal of Experimental & Clinical Cancer Research : CR|January 9, 2025
ONC213: a novel strategy to resensitize resistant AML cells to venetoclax through induction of mitochondrial stressJenna L Carter, Yongwei Su, Eman T Al-Antary, et al.Cancer Research|December 8, 2016
An Essential Role of Maspin in Embryogenesis and Tumor SuppressionSijana H Dzinic, M Margarida Bernardo, Xiaohua Li, et al.Oncotarget|November 7, 2014
Maspin expression in prostate tumor elicits host anti-tumor immunitySijana H Dzinic, Kang Chen, Archana Thakur, et al.Biochemical Pharmacology|February 15, 2026
Loss of cystathionine-β-synthase contributes to elevated OXPHOS, a vulnerability in ara-C-resistant myeloid Leukemia in Down syndromeJenna Thibodeau, Jianlei Zhao, Holly Edwards, et al.Molecular Cancer Therapeutics|July 11, 2019
Novel Pyrrolo[3,2-d]pyrimidine Compounds Target Mitochondrial and Cytosolic One-carbon Metabolism with Broad-spectrum Antitumor EfficacyAamod S Dekhne, Khushbu Shah, Gregory S Ducker, et al.Research Square|May 10, 2023
Enhancing anti-AML activity of venetoclax by isoflavone ME-344 through suppression of OXPHOS and/or purine biosynthesisKatie H Hurrish, Yongwei Su, Shraddha Patel, et al.Biochemical Pharmacology|December 11, 2023
Enhancing anti-AML activity of venetoclax by isoflavone ME-344 through suppression of OXPHOS and/or purine biosynthesis in vitroKatie H Hurrish, Yongwei Su, Shraddha Patel, et al.Cancer Research|January 24, 2024
The Imipridone ONC213 Targets α-Ketoglutarate Dehydrogenase to Induce Mitochondrial Stress and Suppress Oxidative Phosphorylation in Acute Myeloid LeukemiaYongwei Su, Jenna L Carter, Xinyu Li, et al.Pageof 3