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Silvia Gines

Showing results (1-10 of 11) with videos related to

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Neurobiology of Disease|January 14, 2020
Mitochondrial fission in Huntington's disease mouse striatum disrupts ER-mitochondria contacts leading to disturbances in Ca<sup>2+</sup> efflux and Reactive Oxygen Species (ROS) homeostasisMarta Cherubini, Laura Lopez-Molina, Silvia Gines
BMC Neuroscience|January 15, 2005
Reversal of a full-length mutant huntingtin neuronal cell phenotype by chemical inhibitors of polyglutamine-mediated aggregationJin Wang, Silvia Gines, Marcy E MacDonald, et al.
Neuromolecular Medicine|October 7, 2003
Huntington's diseaseMarcy E MacDonald, Silvia Gines, James F Gusella, et al.
The Journal of Biological Chemistry|October 3, 2003
Enhanced Akt signaling is an early pro-survival response that reflects N-methyl-D-aspartate receptor activation in Huntington's disease knock-in striatal cellsSilvia Gines, Elena Ivanova, Ihn-Sik Seong, et al.
Plos One|December 3, 2019
CD200 is up-regulated in R6/1 transgenic mouse model of Huntington's diseaseAndrea Comella Bolla, Tony Valente, Andres Miguez, et al.
Neurobiology of Disease|April 20, 2005
A human single-chain Fv intrabody preferentially targets amino-terminal Huntingtin's fragments in striatal models of Huntington's diseaseTodd W Miller, Chun Zhou, Silvia Gines, et al.
Human Molecular Genetics|February 18, 2003
Specific progressive cAMP reduction implicates energy deficit in presymptomatic Huntington's disease knock-in miceSilvia Gines, Ihn Sik Seong, Elisa Fossale, et al.
Cell Communication and Signaling : CCS|July 16, 2025
Mitochondria from huntington´s disease striatal astrocytes are hypermetabolic and compromise neuronal branchingLaura López-Molina, Alba Pereda-Velarde, Nadia di Franco, et al.
Neurobiology of Disease|January 31, 2006
Decreased association of the transcription factor Sp1 with genes downregulated in Huntington's diseaseAlice S Chen-Plotkin, Ghazaleh Sadri-Vakili, George J Yohrling, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 23, 2017
Phosphorylation of huntingtin at residue T3 is decreased in Huntington's disease and modulates mutant huntingtin protein conformationCristina Cariulo, Lucia Azzollini, Margherita Verani, et al.
Pageof 2

Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
Neurobiology of Disease|January 14, 2020
Mitochondrial fission in Huntington's disease mouse striatum disrupts ER-mitochondria contacts leading to disturbances in Ca<sup>2+</sup> efflux and Reactive Oxygen Species (ROS) homeostasisMarta Cherubini, Laura Lopez-Molina, Silvia Gines
BMC Neuroscience|January 15, 2005
Reversal of a full-length mutant huntingtin neuronal cell phenotype by chemical inhibitors of polyglutamine-mediated aggregationJin Wang, Silvia Gines, Marcy E MacDonald, et al.
Neuromolecular Medicine|October 7, 2003
Huntington's diseaseMarcy E MacDonald, Silvia Gines, James F Gusella, et al.
The Journal of Biological Chemistry|October 3, 2003
Enhanced Akt signaling is an early pro-survival response that reflects N-methyl-D-aspartate receptor activation in Huntington's disease knock-in striatal cellsSilvia Gines, Elena Ivanova, Ihn-Sik Seong, et al.
Plos One|December 3, 2019
CD200 is up-regulated in R6/1 transgenic mouse model of Huntington's diseaseAndrea Comella Bolla, Tony Valente, Andres Miguez, et al.
Neurobiology of Disease|April 20, 2005
A human single-chain Fv intrabody preferentially targets amino-terminal Huntingtin's fragments in striatal models of Huntington's diseaseTodd W Miller, Chun Zhou, Silvia Gines, et al.
Human Molecular Genetics|February 18, 2003
Specific progressive cAMP reduction implicates energy deficit in presymptomatic Huntington's disease knock-in miceSilvia Gines, Ihn Sik Seong, Elisa Fossale, et al.
Cell Communication and Signaling : CCS|July 16, 2025
Mitochondria from huntington´s disease striatal astrocytes are hypermetabolic and compromise neuronal branchingLaura López-Molina, Alba Pereda-Velarde, Nadia di Franco, et al.
Neurobiology of Disease|January 31, 2006
Decreased association of the transcription factor Sp1 with genes downregulated in Huntington's diseaseAlice S Chen-Plotkin, Ghazaleh Sadri-Vakili, George J Yohrling, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 23, 2017
Phosphorylation of huntingtin at residue T3 is decreased in Huntington's disease and modulates mutant huntingtin protein conformationCristina Cariulo, Lucia Azzollini, Margherita Verani, et al.
Pageof 2