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Blood|August 12, 2004
PU.1 is a suppressor of myeloid leukemia, inactivated in mice by gene deletion and mutation of its DNA binding domainWendy D Cook, Benjamin J McCaw, Christopher Herring, et al.
Scientific Reports|February 7, 2020
Author Correction: KCC1 Activation protects Mice from the Development of Experimental Cerebral MalariaElinor Hortle, Lora Starrs, Fiona C Brown, et al.
Scientific Reports|April 25, 2019
KCC1 Activation protects Mice from the Development of Experimental Cerebral MalariaElinor Hortle, Lora Starrs, Fiona C Brown, et al.
Protein Engineering, Design & Selection : PEDS|September 4, 2012
Disulfide linkage engineering for improving biophysical properties of human VH domainsDae Young Kim, Hiba Kandalaft, Wen Ding, et al.
The Pharmacogenomics Journal|November 26, 2021
Pharmacogenomic analysis of a genetically distinct Indigenous populationArvind Jaya Shankar, Sudhir Jadhao, Wendy Hoy, et al.
Infection and Immunity|July 16, 2002
Mice that are congenic for the char2 locus are susceptible to malariaRachel A Burt, Vikki M Marshall, Jamie Wagglen, et al.
Human Genetics|May 11, 2005
Identifying nineteenth century genealogical links from genotypesJim Stankovich, Melanie Bahlo, Justin P Rubio, et al.
Human Genetics|January 11, 2003
Analysis of 15 primary open-angle glaucoma families from Australia identifies a founder effect for the Q368STOP mutation of myocilinPaul N Baird, Jamie E Craig, Andrea J Richardson, et al.
Plos One|June 23, 2012
A novel ENU-mutation in ankyrin-1 disrupts malaria parasite maturation in red blood cells of miceAndreas Greth, Shelley Lampkin, Preethi Mayura-Guru, et al.
Blood|December 1, 2010
Anticipation in familial hematologic malignanciesElizabeth M Tegg, Russell J Thomson, Jim M Stankovich, et al.
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