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Cell|April 9, 2008
RyR1 S-nitrosylation underlies environmental heat stroke and sudden death in Y522S RyR1 knockin miceWilliam J Durham, Paula Aracena-Parks, Cheng Long, et al.
Elife|March 17, 2023
Ablation of palladin in adult heart causes dilated cardiomyopathy associated with intercalated disc abnormalitiesGiuseppina Mastrototaro, Pierluigi Carullo, Jianlin Zhang, et al.
Cell Death & Disease|February 14, 2025
Creatine transporter (SLC6A8) knockout mice exhibit reduced muscle performance, disrupted mitochondrial Ca2+ homeostasis, and severe muscle atrophyIrene Pertici, Donato D'Angelo, Denis Vecellio Reane, et al.
Cell Metabolism|December 3, 2008
Skeletal muscle is a primary target of SOD1G93A-mediated toxicityGabriella Dobrowolny, Michela Aucello, Emanuele Rizzuto, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 16, 2007
An Ryr1I4895T mutation abolishes Ca2+ release channel function and delays development in homozygous offspring of a mutant mouse lineElena Zvaritch, Frederic Depreux, Natasha Kraeva, et al.
European Journal of Translational Myology|February 26, 2016
Persistent Muscle Fiber Regeneration in Long Term Denervation. Past, Present, FutureUgo Carraro, Simona Boncompagni, Valerio Gobbo, et al.
Frontiers in Endocrinology|January 20, 2022
Effects of Titanium Dioxide Nanoparticles on Porcine Prepubertal Sertoli Cells: An "In Vitro" StudyFrancesca Mancuso, Iva Arato, Alessandro Di Michele, et al.
Nature Communications|June 14, 2019
DRP1-mediated mitochondrial shape controls calcium homeostasis and muscle massGiulia Favaro, Vanina Romanello, Tatiana Varanita, et al.
Journal of Neuropathology and Experimental Neurology|November 17, 2009
A subpopulation of rat muscle fibers maintains an assessable excitation-contraction coupling mechanism after long-standing denervation despite lost contractilityRoberta Squecco, Ugo Carraro, Helmut Kern, et al.
Circulation|June 4, 2014
Single delivery of an adeno-associated viral construct to transfer the CASQ2 gene to knock-in mice affected by catecholaminergic polymorphic ventricular tachycardia is able to cure the disease from birth to advanced ageMarco Denegri, Rossana Bongianino, Francesco Lodola, et al.
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