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Kidney International|February 27, 2009
Protein load impairs factor H binding promoting complement-dependent dysfunction of proximal tubular cellsSimona Buelli, Mauro Abbate, Marina Morigi, et al.
The American Journal of Pathology|March 28, 2006
Permselective dysfunction of podocyte-podocyte contact upon angiotensin II unravels the molecular target for renoprotective interventionDaniela Macconi, Mauro Abbate, Marina Morigi, et al.
JCI Insight|August 10, 2018
SGLT2 inhibitor dapagliflozin limits podocyte damage in proteinuric nondiabetic nephropathyPaola Cassis, Monica Locatelli, Domenico Cerullo, et al.
JCI Insight|March 13, 2020
C3a receptor blockade protects podocytes from injury in diabetic nephropathyMarina Morigi, Luca Perico, Daniela Corna, et al.
Nephron|December 23, 2014
Effects of MCP-1 inhibition by bindarit therapy in a rat model of polycystic kidney diseaseCarlamaria Zoja, Daniela Corna, Monica Locatelli, et al.
Journal of the American Society of Nephrology : JASN|September 30, 2003
Protein overload induces fractalkine upregulation in proximal tubular cells through nuclear factor kappaB- and p38 mitogen-activated protein kinase-dependent pathwaysRoberta Donadelli, Cristina Zanchi, Marina Morigi, et al.
The American Journal of Pathology|May 28, 2013
Angiotensin II contributes to diabetic renal dysfunction in rodents and humans via Notch1/Snail pathwayElena Gagliardini, Norberto Perico, Paola Rizzo, et al.
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation|June 11, 2011
Membranous nephropathy associated with IgG4-related diseasePaolo Cravedi, Mauro Abbate, Elena Gagliardini, et al.
Scientific Reports|June 28, 2016
A previously unrecognized role of C3a in proteinuric progressive nephropathyMarina Morigi, Monica Locatelli, Cinzia Rota, et al.
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