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Journal of the American Society of Nephrology : JASN|March 1, 2014
Shiga toxin promotes podocyte injury in experimental hemolytic uremic syndrome via activation of the alternative pathway of complementMonica Locatelli, Simona Buelli, Anna Pezzotta, et al.Stem Cell Research & Therapy|February 12, 2026
NAMPT overexpression enhances the regenerative potential of mesenchymal stromal cell-derived extracellular vesicles in experimental AKISimona Buelli, Michelle Prioli Miranda Soares, Anna Pezzotta, et al.Cells|June 10, 2022
Shiga Toxin 2 Triggers C3a-Dependent Glomerular and Tubular Injury through Mitochondrial Dysfunction in Hemolytic Uremic SyndromeSimona Buelli, Monica Locatelli, Claudia Elisa Carminati, et al.Journal of Immunology (Baltimore, Md. : 1950)|July 9, 2008
Fractalkine and CX3CR1 mediate leukocyte capture by endothelium in response to Shiga toxinCristina Zanchi, Carla Zoja, Marina Morigi, et al.Journal of the American Society of Nephrology : JASN|December 19, 2008
Proteasomal processing of albumin by renal dendritic cells generates antigenic peptidesDaniela Macconi, Chiara Chiabrando, Silvia Schiarea, et al.Journal of Immunology (Baltimore, Md. : 1950)|June 7, 2011
Alternative pathway activation of complement by Shiga toxin promotes exuberant C3a formation that triggers microvascular thrombosisMarina Morigi, Miriam Galbusera, Sara Gastoldi, et al.Journal of the American Society of Nephrology : JASN|December 28, 2013
β-arrestin-1 drives endothelin-1-mediated podocyte activation and sustains renal injurySimona Buelli, Laura Rosanò, Elena Gagliardini, et al.Ebiomedicine|July 3, 2015
Mitochondrial-dependent Autoimmunity in Membranous Nephropathy of IgG4-related DiseaseSimona Buelli, Luca Perico, Miriam Galbusera, et al.The New England Journal of Medicine|July 16, 2011
MYO1E mutations and childhood familial focal segmental glomerulosclerosisCaterina Mele, Paraskevas Iatropoulos, Roberta Donadelli, et al.Pageof 3