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ACS Chemical Biology|August 31, 2017
BNANC Gapmers Revert Splicing and Reduce RNA Foci with Low Toxicity in Myotonic Dystrophy CellsKassie S Manning, Ashish N Rao, Miguel Castro, et al.
Cell Reports|November 17, 2016
Alternative Splicing of Four Trafficking Genes Regulates Myofiber Structure and Skeletal Muscle PhysiologyJimena Giudice, James A Loehr, George G Rodney, et al.
JCI Insight|March 19, 2026
Progressive cardiac phenotypes and reduced reversibility from long-term CUGexp RNA expression in a DM1 mouse modelRong-Chi Hu, Mohammadreza Tabary, Xander Ht Wehrens, et al.
Cell Reports|July 5, 2018
Rbfox-Splicing Factors Maintain Skeletal Muscle Mass by Regulating Calpain3 and ProteostasisRavi K Singh, Arseniy M Kolonin, Marta L Fiorotto, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|April 15, 2005
Dynamic balance between activation and repression regulates pre-mRNA alternative splicing during heart developmentAndrea N Ladd, Myrna G Stenberg, Maurice S Swanson, et al.
Life Science Alliance|March 28, 2023
The role of Limch1 alternative splicing in skeletal muscle functionMatthew S Penna, Rong-Chi Hu, George G Rodney, et al.
RNA (New York, N.Y.)|March 22, 2003
Antagonistic regulation of alpha-actinin alternative splicing by CELF proteins and polypyrimidine tract binding proteinNatalia Gromak, Arianne J Matlin, Thomas A Cooper, et al.
Human Molecular Genetics|May 18, 2018
Mechanisms of skeletal muscle wasting in a mouse model for myotonic dystrophy type 1Ginny R Morriss, Kimal Rajapakshe, Shixia Huang, et al.
Nucleic Acids Research|January 9, 2023
Alternative splicing mediates the compensatory upregulation of MBNL2 upon MBNL1 loss-of-functionLarissa Nitschke, Rong-Chi Hu, Andrew N Miller, et al.
Molecular and Cellular Biology|July 1, 2005
Cardiac tissue-specific repression of CELF activity disrupts alternative splicing and causes cardiomyopathyAndrea N Ladd, George Taffet, Craig Hartley, et al.
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