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Human Molecular Genetics|July 7, 2010
CUGBP1 overexpression in mouse skeletal muscle reproduces features of myotonic dystrophy type 1Amanda J Ward, Mendell Rimer, James M Killian, et al.
The Journal of Clinical Investigation|September 8, 2007
Elevation of RNA-binding protein CUGBP1 is an early event in an inducible heart-specific mouse model of myotonic dystrophyGuey-Shin Wang, Debra L Kearney, Mariella De Biasi, et al.
Human Molecular Genetics|January 7, 2010
Heart-specific overexpression of CUGBP1 reproduces functional and molecular abnormalities of myotonic dystrophy type 1Misha Koshelev, Satyam Sarma, Roger E Price, et al.
Nucleic Acids Research|September 17, 2013
The splicing landscape is globally reprogrammed during male meiosisRalf Schmid, Sushma Nagaraja Grellscheid, Ingrid Ehrmann, et al.
Iscience|October 30, 2024
Alternatively spliced MAP4 isoforms have key roles in maintaining microtubule organization and skeletal muscle functionLathan Lucas, Larissa Nitschke, Brandon Nguyen, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2008
Expanded CTG repeats within the DMPK 3' UTR causes severe skeletal muscle wasting in an inducible mouse model for myotonic dystrophyJames P Orengo, Pierre Chambon, Daniel Metzger, et al.
The International Journal of Biochemistry & Cell Biology|October 15, 2018
Modulation of alternative splicing of trafficking genes by genome editing reveals functional consequences in muscle biologyR Eric Blue, Amrita Koushik, Nichlas M Engels, et al.
Cell Reports|January 14, 2014
The Mef2 transcription network is disrupted in myotonic dystrophy heart tissue, dramatically altering miRNA and mRNA expressionAuinash Kalsotra, Ravi K Singh, Priyatansh Gurha, et al.
Human Molecular Genetics|August 30, 2007
Expression, localization and tau exon 10 splicing activity of the brain RNA-binding protein TNRC4J Paul Chapple, Karen Anthony, Teresa Rodriguez Martin, et al.
Elife|August 23, 2017
Extensive alternative splicing transitions during postnatal skeletal muscle development are required for calcium handling functionsAmy E Brinegar, Zheng Xia, James Anthony Loehr, et al.
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