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Sirenko

Showing results (221-230 of 300) with videos related to

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Wiadomosci Lekarskie (Warsaw, Poland : 1960)|July 18, 2023
DEVELOPMENT OF COORDINATION ABILITIES IN 6-10 YEARS OLD BOYS WITH POSTURAL DISORDERSOleksii V Tymoshenko, Zhanna H Domina, Valerii V Trotsenko, et al.
Archives of Biochemistry and Biophysics|March 7, 2018
The primary cause of muscle disfunction associated with substitutions E240K and R244G in tropomyosin is aberrant behavior of tropomyosin and response of actin and myosin during ATPase cycleArmen O Simonyan, Vladimir V Sirenko, Olga E Karpicheva, et al.
Chemistry (Weinheim an Der Bergstrasse, Germany)|May 10, 2022
Four-Step Spin Crossover in a New Cyano-Bridged Iron-Silver Coordination PolymerOlesia I Kucheriv, Sergii I Shylin, Valerii Y Sirenko, et al.
Kardiologiia|February 1, 1991
[Possibilities of computerized bidimensional echocardiography in the diagnosis of unstable angina pectoris]I K Sledzevskaia, V K Tashchuk, V A Shumakov, et al.
Likars'Ka Sprava|September 1, 1993
[The complex assessment of myocardial electrical instability in exacerbated ischemic heart disease]V A Bobrov, I E Malinovskaia, V K Tashchuk, et al.
International Journal of Molecular Sciences|December 15, 2018
The Primary Causes of Muscle Dysfunction Associated with the Point Mutations in Tpm3.12; Conformational Analysis of Mutant Proteins as a Tool for Classification of MyopathiesYurii S Borovikov, Olga E Karpicheva, Armen O Simonyan, et al.
Likars'Ka Sprava|September 1, 1994
[The structure of the ventricular arrhythmias in patients in the first 24 hours of a myocardial infarct]V A Bobrov, I E Malinovskaia, V K Tashchuk, et al.
Biochemical and Biophysical Research Communications|May 25, 2018
The reason for the low Ca<sup>2+</sup>-sensitivity of thin filaments associated with the Glu41Lys mutation in the TPM2 gene is "freezing" of tropomyosin near the outer domain of actin and inhibition of actin monomer switching off during the ATPase cycleStanislava V Avrova, Olga E Karpicheva, Nikita A Rysev, et al.
Journal of Molecular and Cellular Cardiology|November 27, 2013
Sarcoplasmic reticulum Ca2+ cycling protein phosphorylation in a physiologic Ca2+ milieu unleashes a high-power, rhythmic Ca2+ clock in ventricular myocytes: relevance to arrhythmias and bio-pacemaker designSyevda Sirenko, Victor A Maltsev, Larissa A Maltseva, et al.
Journal of Molecular and Cellular Cardiology|September 27, 2014
Stochasticity intrinsic to coupled-clock mechanisms underlies beat-to-beat variability of spontaneous action potential firing in sinoatrial node pacemaker cellsYael Yaniv, Alexey E Lyashkov, Syevda Sirenko, et al.
Pageof 30

Showing results (221-230 of 300) with videos related to

Sort By:
Pageof 30
Wiadomosci Lekarskie (Warsaw, Poland : 1960)|July 18, 2023
DEVELOPMENT OF COORDINATION ABILITIES IN 6-10 YEARS OLD BOYS WITH POSTURAL DISORDERSOleksii V Tymoshenko, Zhanna H Domina, Valerii V Trotsenko, et al.
Archives of Biochemistry and Biophysics|March 7, 2018
The primary cause of muscle disfunction associated with substitutions E240K and R244G in tropomyosin is aberrant behavior of tropomyosin and response of actin and myosin during ATPase cycleArmen O Simonyan, Vladimir V Sirenko, Olga E Karpicheva, et al.
Chemistry (Weinheim an Der Bergstrasse, Germany)|May 10, 2022
Four-Step Spin Crossover in a New Cyano-Bridged Iron-Silver Coordination PolymerOlesia I Kucheriv, Sergii I Shylin, Valerii Y Sirenko, et al.
Kardiologiia|February 1, 1991
[Possibilities of computerized bidimensional echocardiography in the diagnosis of unstable angina pectoris]I K Sledzevskaia, V K Tashchuk, V A Shumakov, et al.
Likars'Ka Sprava|September 1, 1993
[The complex assessment of myocardial electrical instability in exacerbated ischemic heart disease]V A Bobrov, I E Malinovskaia, V K Tashchuk, et al.
International Journal of Molecular Sciences|December 15, 2018
The Primary Causes of Muscle Dysfunction Associated with the Point Mutations in Tpm3.12; Conformational Analysis of Mutant Proteins as a Tool for Classification of MyopathiesYurii S Borovikov, Olga E Karpicheva, Armen O Simonyan, et al.
Likars'Ka Sprava|September 1, 1994
[The structure of the ventricular arrhythmias in patients in the first 24 hours of a myocardial infarct]V A Bobrov, I E Malinovskaia, V K Tashchuk, et al.
Biochemical and Biophysical Research Communications|May 25, 2018
The reason for the low Ca<sup>2+</sup>-sensitivity of thin filaments associated with the Glu41Lys mutation in the TPM2 gene is "freezing" of tropomyosin near the outer domain of actin and inhibition of actin monomer switching off during the ATPase cycleStanislava V Avrova, Olga E Karpicheva, Nikita A Rysev, et al.
Journal of Molecular and Cellular Cardiology|November 27, 2013
Sarcoplasmic reticulum Ca2+ cycling protein phosphorylation in a physiologic Ca2+ milieu unleashes a high-power, rhythmic Ca2+ clock in ventricular myocytes: relevance to arrhythmias and bio-pacemaker designSyevda Sirenko, Victor A Maltsev, Larissa A Maltseva, et al.
Journal of Molecular and Cellular Cardiology|September 27, 2014
Stochasticity intrinsic to coupled-clock mechanisms underlies beat-to-beat variability of spontaneous action potential firing in sinoatrial node pacemaker cellsYael Yaniv, Alexey E Lyashkov, Syevda Sirenko, et al.
Pageof 30