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Redox Biology|October 7, 2021
Nucleus-mitochondria positive feedback loop formed by ERK5 S496 phosphorylation-mediated poly (ADP-ribose) polymerase activation provokes persistent pro-inflammatory senescent phenotype and accelerates coronary atherosclerosis after chemo-radiationSivareddy Kotla, Aijun Zhang, Masaki Imanishi, et al.
Circulation Research|June 2, 2023
An ERK5-NRF2 Axis Mediates Senescence-Associated Stemness and AtherosclerosisJun-Ichi Abe, Masaki Imanishi, Shengyu Li, et al.
Frontiers in Cardiovascular Medicine|October 2, 2023
Premature senescence and cardiovascular disease following cancer treatments: mechanistic insightsAshita Jain, Diego Casanova, Alejandra Valdivia Padilla, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|January 16, 2025
Reevaluating Anti-Inflammatory Therapy: Targeting Senescence to Balance Anti-Cancer Efficacy and Vascular DiseaseBernardo Casso-Chapa, Norma Alicia Vazquez González, Nhat-Tu Le, et al.
Biorxiv : the Preprint Server for Biology|July 16, 2025
Downregulation of LATS1/2 Drives Endothelial Senescence-Associated Stemness (SAS) and Atherothrombotic Lesion FormationSivareddy Kotla, Jonghae Lee, Kyung Ae Ko, et al.
Circulation Research|May 27, 2026
LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque ThrombosisSivareddy Kotla, Jonghae Lee, Kyung Ae Ko, et al.
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