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Journal for Immunotherapy of Cancer|May 11, 2026
Humoral contributions to checkpoint blockade therapyMichelle L McKeague, Sokratis A Apostolidis
Cytokine|June 2, 2015
Update on the role of Interleukin 17 in rheumatologic autoimmune diseasesChristine Konya, Ziv Paz, Sokratis A Apostolidis, et al.
Trends in Molecular Medicine|July 28, 2016
Empowering Regulatory T Cells in AutoimmunityIsaac R Kasper, Sokratis A Apostolidis, Amir Sharabi, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 13, 2011
Induction of PP2A Bβ, a regulator of IL-2 deprivation-induced T-cell apoptosis, is deficient in systemic lupus erythematosusJosé C Crispín, Sokratis A Apostolidis, Melissa I Finnell, et al.
The Journal of Biological Chemistry|August 7, 2013
Protein phosphatase 2A enables expression of interleukin 17 (IL-17) through chromatin remodelingSokratis A Apostolidis, Thomas Rauen, Christian M Hedrich, et al.
Journal of Interferon & Cytokine Research : the Official Journal of the International Society for Interferon and Cytokine Research|September 1, 2011
The dysregulation of cytokine networks in systemic lupus erythematosusSokratis A Apostolidis, Linda A Lieberman, Katalin Kis-Toth, et al.
Journal of Leukocyte Biology|February 14, 2020
TCR-α/β CD4<sup>-</sup> CD8<sup>-</sup> double negative T cells arise from CD8<sup>+</sup> T cellsNoé Rodríguez-Rodríguez, Giovanna Flores-Mendoza, Sokratis A Apostolidis, et al.
Frontiers in Immunology|October 18, 2018
Single Cell RNA Sequencing Identifies HSPG2 and APLNR as Markers of Endothelial Cell Injury in Systemic Sclerosis SkinSokratis A Apostolidis, Giuseppina Stifano, Tracy Tabib, et al.
Pacific Symposium on Biocomputing. Pacific Symposium on Biocomputing|December 13, 2024
Plasma protein-based and polygenic risk scores serve complementary roles in predicting inflammatory bowel diseaseJakob Woerner, Thomas Westbrook, Seokho Jeong, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 17, 2012
Cutting edge: protein phosphatase 2A confers susceptibility to autoimmune disease through an IL-17-dependent mechanismJosé C Crispín, Sokratis A Apostolidis, Florencia Rosetti, et al.
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