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Bioinformation|May 11, 2026
Expert opinion survey on diagnostic and treatment-planning gaps in orthodontics among dental interns: Validation of the need for a MOOC-based educational interventionTanusha Mahobia, Pallavi Daigavane, Piyush Khandelwal, et al.
Sub-Cellular Biochemistry|February 20, 2026
The Role of the Golgi Apparatus in NeurodegenerationSina Shadfar, Sara Assar Kashani, Shashi Gautam, et al.
Bioorganic & Medicinal Chemistry|March 4, 2016
Efficacy of peptide nucleic acid and selected conjugates against specific cellular pathologies of amyotrophic lateral sclerosisElisse C Browne, Sonam Parakh, Luke F Duncan, et al.
Journal of Molecular Neuroscience : MN|June 13, 2020
Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral SclerosisEmma R Perri, Sonam Parakh, Marta Vidal, et al.
Journal of Molecular Neuroscience : MN|May 24, 2020
The Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral SclerosisEmma R Perri, Sonam Parakh, Marta Vidal, et al.
Neuromolecular Medicine|June 11, 2024
Protein Disulfide Isomerase Endoplasmic Reticulum Protein 57 (ERp57) is Protective Against ALS-Associated Mutant TDP-43 in Neuronal CellsSonam Parakh, Emma R Perri, Marta Vidal, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|May 16, 2025
Pathological forms of TDP-43 in amyotrophic lateral sclerosis (ALS) promote aberrant telomere elongationAnna Konopka, Md Shafi Jamali, Megan Fowler, et al.
Acta Neuropathologica|August 24, 2015
Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALSKai Y Soo, Mark Halloran, Vinod Sundaramoorthy, et al.
Plos One|December 7, 2013
ALS-associated TDP-43 induces endoplasmic reticulum stress, which drives cytoplasmic TDP-43 accumulation and stress granule formationAdam K Walker, Kai Y Soo, Vinod Sundaramoorthy, et al.
Cellular and Molecular Life Sciences : CMLS|December 6, 2019
Amyotrophic lateral sclerosis-linked UBQLN2 mutants inhibit endoplasmic reticulum to Golgi transport, leading to Golgi fragmentation and ER stressMark Halloran, Audrey M G Ragagnin, Marta Vidal, et al.
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