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Free Radical Biology & Medicine|March 12, 2025
Mitochondrial ACSS1-K635 acetylation knock-in mice exhibit altered liver lipid metabolism on a ketogenic dietGuogang Xu, Joseph Schell, Songhua Quan, et al.Journal of Medicinal Chemistry|February 13, 2026
Development of DOT1L-Targeted Protein Degraders for Treating MLL-r LeukemiaSonghua Quan, Kenji Unno, Dikshat G Gupta, et al.Cancer Research|July 3, 2024
Asparagine Dependency Is a Targetable Metabolic Vulnerability in TP53-Altered Castration-Resistant Prostate CancerYoung A Yoo, Songhua Quan, William Yang, et al.Nature Communications|June 5, 2019
Lysine 68 acetylation directs MnSOD as a tetrameric detoxification complex versus a monomeric tumor promoterYueming Zhu, Xianghui Zou, Angela E Dean, et al.Nature Communications|January 8, 2024
Mechanism-centric regulatory network identifies NME2 and MYC programs as markers of Enzalutamide resistance in CRPCSukanya Panja, Mihai Ioan Truica, Christina Y Yu, et al.Science Advances|July 16, 2025
Impaired mitochondrial metabolism is a critical cancer vulnerability for MYC inhibitorsWilliam Yang, Qianyu Guo, Songhua Quan, et al.Science Advances|May 17, 2024
Mitochondrial ACSS1-K635 acetylation knock-in mice exhibit altered metabolism, cell senescence, and nonalcoholic fatty liver diseaseGuogang Xu, Songhua Quan, Joseph Schell, et al.Biorxiv : the Preprint Server for Biology|March 18, 2026
A bivalent lysine-acetylated small-molecule binding site in MYCDikshat G Gupta, Mihai I Truica, Adam W T Steffeck, et al.Pageof 2