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Sonja Stahl

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Developmental Dynamics : an Official Publication of the American Association of Anatomists|December 23, 2004
Non-heparan sulfate-binding interactions of endostatin/collagen XVIII in murine developmentNatalia Rychkova, Sonja Stahl, Sabine Gaetzner, et al.
Journal of Negative Results in Biomedicine|June 2, 2006
VEGF receptors on PC12 cells mediate transient activation of ERK1/2 and Akt: comparison of nerve growth factor and vascular endothelial growth factorIngrid Berger, Sonja Stahl, Natalia Rychkova, et al.
Human Molecular Genetics|December 18, 2008
A two-hit mechanism causes cerebral cavernous malformations: complete inactivation of CCM1, CCM2 or CCM3 in affected endothelial cellsAxel Pagenstecher, Sonja Stahl, Ulrich Sure, et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|August 24, 2005
Endostatin phenylalanines 31 and 34 define a receptor binding siteSonja Stahl, Sabine Gaetzner, Thomas D Mueller, et al.
Neurogenetics|September 24, 2010
Evidence for anti-angiogenic and pro-survival functions of the cerebral cavernous malformation protein 3Elisa Schleider, Sonja Stahl, Joycelyn Wüstehube, et al.
Child'S Nervous System : Chns : Official Journal of the International Society for Pediatric Neurosurgery|September 20, 2006
Clinical impact of CCM mutation detection in familial cavernous angiomaOguzkan Sürücü, Ulrich Sure, Sabine Gaetzner, et al.
Biochimica Et Biophysica Acta|September 4, 2007
Proteomic analysis of cathepsin B- and L-deficient mouse brain lysosomesSonja Stahl, Yvonne Reinders, Esther Asan, et al.
Neurogenetics|July 28, 2007
CCM3 interacts with CCM2 indicating common pathogenesis for cerebral cavernous malformationsKatrin Voss, Sonja Stahl, Elisa Schleider, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|February 8, 2005
Endostatin's heparan sulfate-binding site is essential for inhibition of angiogenesis and enhances in situ binding to capillary-like structures in bone explantsSabine Gaetzner, Martine M L Deckers, Sonja Stahl, et al.
Human Mutation|April 17, 2009
Functional analyses of human and zebrafish 18-amino acid in-frame deletion pave the way for domain mapping of the cerebral cavernous malformation 3 proteinKatrin Voss, Sonja Stahl, Benjamin M Hogan, et al.
Pageof 2

Showing results (1-10 of 12) with videos related to

Sort By:
Pageof 2
Developmental Dynamics : an Official Publication of the American Association of Anatomists|December 23, 2004
Non-heparan sulfate-binding interactions of endostatin/collagen XVIII in murine developmentNatalia Rychkova, Sonja Stahl, Sabine Gaetzner, et al.
Journal of Negative Results in Biomedicine|June 2, 2006
VEGF receptors on PC12 cells mediate transient activation of ERK1/2 and Akt: comparison of nerve growth factor and vascular endothelial growth factorIngrid Berger, Sonja Stahl, Natalia Rychkova, et al.
Human Molecular Genetics|December 18, 2008
A two-hit mechanism causes cerebral cavernous malformations: complete inactivation of CCM1, CCM2 or CCM3 in affected endothelial cellsAxel Pagenstecher, Sonja Stahl, Ulrich Sure, et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|August 24, 2005
Endostatin phenylalanines 31 and 34 define a receptor binding siteSonja Stahl, Sabine Gaetzner, Thomas D Mueller, et al.
Neurogenetics|September 24, 2010
Evidence for anti-angiogenic and pro-survival functions of the cerebral cavernous malformation protein 3Elisa Schleider, Sonja Stahl, Joycelyn Wüstehube, et al.
Child'S Nervous System : Chns : Official Journal of the International Society for Pediatric Neurosurgery|September 20, 2006
Clinical impact of CCM mutation detection in familial cavernous angiomaOguzkan Sürücü, Ulrich Sure, Sabine Gaetzner, et al.
Biochimica Et Biophysica Acta|September 4, 2007
Proteomic analysis of cathepsin B- and L-deficient mouse brain lysosomesSonja Stahl, Yvonne Reinders, Esther Asan, et al.
Neurogenetics|July 28, 2007
CCM3 interacts with CCM2 indicating common pathogenesis for cerebral cavernous malformationsKatrin Voss, Sonja Stahl, Elisa Schleider, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|February 8, 2005
Endostatin's heparan sulfate-binding site is essential for inhibition of angiogenesis and enhances in situ binding to capillary-like structures in bone explantsSabine Gaetzner, Martine M L Deckers, Sonja Stahl, et al.
Human Mutation|April 17, 2009
Functional analyses of human and zebrafish 18-amino acid in-frame deletion pave the way for domain mapping of the cerebral cavernous malformation 3 proteinKatrin Voss, Sonja Stahl, Benjamin M Hogan, et al.
Pageof 2