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Blood|February 16, 2011
Lenalidomide targets clonogenic side population in multiple myeloma: pathophysiologic and clinical implicationsJana Jakubikova, Sophia Adamia, Maria Kost-Alimova, et al.
Blood|October 22, 2010
MicroRNAs 15a/16-1 function as tumor suppressor genes in multiple myelomaMoshe E Gatt, Jian-Jun Zhao, Margaret S Ebert, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|May 13, 2015
APO866 Increases Antitumor Activity of Cyclosporin-A by Inducing Mitochondrial and Endoplasmic Reticulum Stress in Leukemia CellsAntonia Cagnetta, Irene Caffa, Chirag Acharya, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|November 29, 2013
A genome-wide aberrant RNA splicing in patients with acute myeloid leukemia identifies novel potential disease markers and therapeutic targetsSophia Adamia, Benjamin Haibe-Kains, Patrick M Pilarski, et al.
British Journal of Haematology|February 18, 2009
Expression of regulatory genes for lymphoplasmacytic cell differentiation in Waldenstrom MacroglobulinemiaXavier Leleu, Zachary R Hunter, Lian Xu, et al.
Blood|December 18, 2015
Evidence for a role of the histone deacetylase SIRT6 in DNA damage response of multiple myeloma cellsMichele Cea, Antonia Cagnetta, Sophia Adamia, et al.
Blood|March 20, 2010
Discovery of a small-molecule type II inhibitor of wild-type and gatekeeper mutants of BCR-ABL, PDGFRalpha, Kit, and Src kinases: novel type II inhibitor of gatekeeper mutantsEllen Weisberg, Hwan Geun Choi, Arghya Ray, et al.
Blood|February 28, 2014
NOTCH2 and FLT3 gene mis-splicings are common events in patients with acute myeloid leukemia (AML): new potential targets in AMLSophia Adamia, Michal Bar-Natan, Benjamin Haibe-Kains, et al.
Cancer Cell|November 20, 2020
Reduced Mitochondrial Apoptotic Priming Drives Resistance to BH3 Mimetics in Acute Myeloid LeukemiaShruti Bhatt, Marissa S Pioso, Elyse Anne Olesinski, et al.
Blood Cancer Discovery|March 5, 2024
Acquired Multidrug Resistance in AML Is Caused by Low Apoptotic Priming in Relapsed MyeloblastsElyse A Olesinski, Karanpreet Singh Bhatia, Chuqi Wang, et al.
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