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Soria

Showing results (521-530 of 6,723) with videos related to

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Revue Scientifique Et Technique (International Office of Epizootics)|September 1, 1992
[Experimental infection of sheep with a rabies virus of canine origin: study of the pathogenicity for that species]R Soria Baltazar, M Artois, J Blancou
Angiogenesis|October 1, 2003
Endothelial metalloprotease-disintegrin protein (ADAM) is implicated in angiogenesis in vitroV Trochon, H Li, M Vasse, et al.
Thrombosis and Haemostasis|March 8, 2003
Insights in the molecular mechanisms of the anti-angiogenic effect of an inhibitor of 3-hydroxy-3-methylglutaryl coenzyme A reductaseLoïc Vincent, Patricia Albanese, Heidi Bompais, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|October 1, 1990
Absence of inhibition by lipoprotein (a) inhibition of tPA induced thrombolysis in a patient's plasma milieuH Lu, J Bruckert, J Soria, et al.
FEBS Letters|December 12, 1994
Blockage of the urokinase receptor on the cell surface: construction and characterization of a hybrid protein consisting of the N-terminal fragment of human urokinase and human albuminH Lu, P Yeh, J D Guitton, et al.
Clinica Chimica Acta; International Journal of Clinical Chemistry|May 20, 2004
Activation of PAF receptor by oxidised LDL in human monocytes stimulates chemokine releases but not urokinase-type plasminogen activator expressionJean-Louis Beaudeux, Toihiri Said, Ewa Ninio, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|July 26, 2002
Oncostatin M induces procoagulant activity in human vascular smooth muscle cells by modulating the balance between tissue factor and tissue factor pathway inhibitorF Mirshahi, M Vasse, A Tedgui, et al.
Annals of the New York Academy of Sciences|July 20, 2001
Fibrinogen: a vascular risk factor, why? Contributing effect of oncostatin M on both fibrinogen biosynthesis by hepatocytes and participation in atherothrombotic risk related to modifications of endothelial cellsF Mirshahi, M Vasse, L Vincent, et al.
Thrombosis and Haemostasis|December 14, 1999
Abnormal fibrin clot architecture in nephrotic patients is related to hypofibrinolysis: influence of plasma biochemical modifications: a possible mechanism for the high thrombotic tendency?J P Colle, Z Mishal, C Lesty, et al.
Biochemical and Biophysical Research Communications|September 9, 2006
Malignant hematopoietic cells induce an increased expression of VEGFR-1 and VEGFR-3 on bone marrow endothelial cells via AKT and mTOR signalling pathwaysP Mirshahi, S K Toprak, A M Faussat, et al.
Pageof 673

Showing results (521-530 of 6,723) with videos related to

Sort By:
Pageof 673
Revue Scientifique Et Technique (International Office of Epizootics)|September 1, 1992
[Experimental infection of sheep with a rabies virus of canine origin: study of the pathogenicity for that species]R Soria Baltazar, M Artois, J Blancou
Angiogenesis|October 1, 2003
Endothelial metalloprotease-disintegrin protein (ADAM) is implicated in angiogenesis in vitroV Trochon, H Li, M Vasse, et al.
Thrombosis and Haemostasis|March 8, 2003
Insights in the molecular mechanisms of the anti-angiogenic effect of an inhibitor of 3-hydroxy-3-methylglutaryl coenzyme A reductaseLoïc Vincent, Patricia Albanese, Heidi Bompais, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|October 1, 1990
Absence of inhibition by lipoprotein (a) inhibition of tPA induced thrombolysis in a patient's plasma milieuH Lu, J Bruckert, J Soria, et al.
FEBS Letters|December 12, 1994
Blockage of the urokinase receptor on the cell surface: construction and characterization of a hybrid protein consisting of the N-terminal fragment of human urokinase and human albuminH Lu, P Yeh, J D Guitton, et al.
Clinica Chimica Acta; International Journal of Clinical Chemistry|May 20, 2004
Activation of PAF receptor by oxidised LDL in human monocytes stimulates chemokine releases but not urokinase-type plasminogen activator expressionJean-Louis Beaudeux, Toihiri Said, Ewa Ninio, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|July 26, 2002
Oncostatin M induces procoagulant activity in human vascular smooth muscle cells by modulating the balance between tissue factor and tissue factor pathway inhibitorF Mirshahi, M Vasse, A Tedgui, et al.
Annals of the New York Academy of Sciences|July 20, 2001
Fibrinogen: a vascular risk factor, why? Contributing effect of oncostatin M on both fibrinogen biosynthesis by hepatocytes and participation in atherothrombotic risk related to modifications of endothelial cellsF Mirshahi, M Vasse, L Vincent, et al.
Thrombosis and Haemostasis|December 14, 1999
Abnormal fibrin clot architecture in nephrotic patients is related to hypofibrinolysis: influence of plasma biochemical modifications: a possible mechanism for the high thrombotic tendency?J P Colle, Z Mishal, C Lesty, et al.
Biochemical and Biophysical Research Communications|September 9, 2006
Malignant hematopoietic cells induce an increased expression of VEGFR-1 and VEGFR-3 on bone marrow endothelial cells via AKT and mTOR signalling pathwaysP Mirshahi, S K Toprak, A M Faussat, et al.
Pageof 673