Showing results (11-20 of 41) with videos related to
Sort By:
Pageof 5
Hypertension (Dallas, Tex. : 1979)|December 25, 2013
Macrophage migration inhibitory factor deletion exacerbates pressure overload-induced cardiac hypertrophy through mitigating autophagyXihui Xu, Yinan Hua, Sreejayan Nair, et al.Molecular Nutrition & Food Research|April 5, 2013
Bisdemethoxycurcumin inhibits PDGF-induced vascular smooth muscle cell motility and proliferationYinan Hua, Julia Dolence, Shalini Ramanan, et al.Recent Patents on Endocrine, Metabolic & Immune Drug Discovery|October 30, 2014
Contribution of ALDH2 polymorphism to alcoholism-associated hypertensionNan Hu, Yingmei Zhang, Sreejayan Nair, et al.Cell Death & Disease|December 6, 2019
Correction: Cardiomyocyte-specific disruption of Cathepsin K protects against doxorubicin-induced cardiotoxicityRui Guo, Yinan Hua, Jun Ren, et al.Cell Death & Disease|June 9, 2018
Cardiomyocyte-specific disruption of Cathepsin K protects against doxorubicin-induced cardiotoxicityRui Guo, Yinan Hua, Jun Ren, et al.Biochimica Et Biophysica Acta. Molecular Basis of Disease|September 25, 2017
Deletion of protein tyrosine phosphatase 1B obliterates endoplasmic reticulum stress-induced myocardial dysfunction through regulation of autophagyShuyi Wang, Xiyao Chen, Sreejayan Nair, et al.Toxicological Sciences : an Official Journal of the Society of Toxicology|August 6, 2014
AMP-activated protein kinase deficiency rescues paraquat-induced cardiac contractile dysfunction through an autophagy-dependent mechanismQiurong Wang, Lifang Yang, Yinan Hua, et al.Drug Design, Development and Therapy|December 7, 2016
CARD9 as a potential target in cardiovascular diseaseMatthew R Peterson, Samantha E Haller, Jun Ren, et al.Scientific Reports|August 20, 2017
Cathepsin K knockout protects against cardiac dysfunction in diabetic miceRui Guo, Yinan Hua, Olivia Rogers, et al.Diabetes|October 17, 2012
Cathepsin K knockout mitigates high-fat diet-induced cardiac hypertrophy and contractile dysfunctionYinan Hua, Yingmei Zhang, Julia Dolence, et al.Pageof 5