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Therapeutic Advances in Neurological Disorders|March 22, 2012
Incidence of therapy-related acute leukaemia in mitoxantrone-treated multiple sclerosis patients in GermanyAnke Stroet, Claudia Hemmelmann, Michaela Starck, et al.Journal of Neuroimmunology|December 4, 2014
Lack of efficacy of mitoxantrone in primary progressive Multiple Sclerosis irrespective of pharmacogenetic factors: a multi-center, retrospective analysisSteffi Grey Née Cotte, Anke Salmen Née Stroet, Nico von Ahsen, et al.The EMBO Journal|November 28, 2015
Gatekeeper role of brain antigen-presenting CD11c+ cells in neuroinflammationMagdalena Paterka, Volker Siffrin, Jan O Voss, et al.Plos One|August 12, 2016
The Relationship between Gray Matter Quantitative MRI and Disability in Secondary Progressive Multiple SclerosisRené-Maxime Gracien, Alina Jurcoane, Marlies Wagner, et al.Clinical Neurology and Neurosurgery|May 11, 2010
Patterns of retinal nerve fiber layer loss in multiple sclerosis patients with or without optic neuritis and glaucoma patientsMarkus Bock, Alexander U Brandt, Jan Dörr, et al.Fortschritte Der Neurologie-Psychiatrie|July 13, 2026
[Neurofilament light chain as a tool for assessing disease activity and treatment response in Multiple Sclerosis]Hernan Inojosa, Katja Akgün, Lars Masanneck, et al.Neurology(R) Neuroimmunology & Neuroinflammation|October 31, 2022
Effect of Estimated Blood Volume and Body Mass Index on GFAP and NfL Levels in the Serum and CSF of Patients With Multiple SclerosisYavor Yalachkov, Jan Hendrik Schäfer, Jasmin Jakob, et al.Journal of Neuro-Oncology|January 25, 2008
The two-pore domain potassium channel TASK3 functionally impacts glioma cell deathSven G Meuth, Alexander M Herrmann, Chi W Ip, et al.Brain : a Journal of Neurology|March 23, 2013
Modulation of dendritic cell properties by laquinimod as a mechanism for modulating multiple sclerosisValérie Jolivel, Felix Luessi, Joumana Masri, et al.Pflugers Archiv : European Journal of Physiology|November 8, 2013
A splice variant of the two-pore domain potassium channel TREK-1 with only one pore domain reduces the surface expression of full-length TREK-1 channelsSusanne Rinné, Vijay Renigunta, Günter Schlichthörl, et al.Pageof 43