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Journal of Virology
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October 29, 2003
Human papillomavirus type 16 E7 oncoprotein can induce abnormal centrosome duplication through a mechanism independent of inactivation of retinoblastoma protein family members
Stefan Duensing, Karl Münger
Neoplasia (New York, N.Y.)
|
September 29, 2012
CAND1 promotes PLK4-mediated centriole overduplication and is frequently disrupted in prostate cancer
Nina Korzeniewski, Markus Hohenfellner, Stefan Duensing
Expert Opinion on Therapeutic Targets
|
October 16, 2012
The centrosome as potential target for cancer therapy and prevention
Nina Korzeniewski, Markus Hohenfellner, Stefan Duensing
Molecular Cancer
|
May 26, 2011
The HPV-16 E7 oncoprotein induces centriole multiplication through deregulation of Polo-like kinase 4 expression
Nina Korzeniewski, Benjamin Treat, Stefan Duensing
Cancer Letters
|
November 16, 2010
Genomic instability and cancer: lessons learned from human papillomaviruses
Nina Korzeniewski, Nicole Spardy, Anette Duensing, et al.
Molecular Biology of the Cell
|
September 24, 2010
Daughter centriole elongation is controlled by proteolysis
Nina Korzeniewski, Rolando Cuevas, Anette Duensing, et al.
Oncogene
|
February 3, 2004
The helix-loop-helix protein ID1 localizes to centrosomes and rapidly induces abnormal centrosome numbers
Jens Hasskarl, Stefan Duensing, Edwin Manuel, et al.
Urologic Oncology
|
October 22, 2013
Human papillomaviruses in urological malignancies: a critical assessment
Yanis Tolstov, Boris Hadaschik, Sascha Pahernik, et al.
Molecular Cancer
|
September 23, 2003
Excessive centrosome abnormalities without ongoing numerical chromosome instability in a Burkitt's lymphoma
Stefan Duensing, Benjamin H Lee, Paola Dal Cin, et al.
Urologic Oncology
|
June 17, 2014
High-risk prostate cancer: a disease of genomic instability
María A Tapia-Laliena, Nina Korzeniewski, Markus Hohenfellner, et al.
Page
of 14
Search research articles
Search
Showing results (11-20 of 139) with videos related to
Sort By:
Page
of 14
Journal of Virology
|
October 29, 2003
Human papillomavirus type 16 E7 oncoprotein can induce abnormal centrosome duplication through a mechanism independent of inactivation of retinoblastoma protein family members
Stefan Duensing, Karl Münger
Neoplasia (New York, N.Y.)
|
September 29, 2012
CAND1 promotes PLK4-mediated centriole overduplication and is frequently disrupted in prostate cancer
Nina Korzeniewski, Markus Hohenfellner, Stefan Duensing
Expert Opinion on Therapeutic Targets
|
October 16, 2012
The centrosome as potential target for cancer therapy and prevention
Nina Korzeniewski, Markus Hohenfellner, Stefan Duensing
Molecular Cancer
|
May 26, 2011
The HPV-16 E7 oncoprotein induces centriole multiplication through deregulation of Polo-like kinase 4 expression
Nina Korzeniewski, Benjamin Treat, Stefan Duensing
Cancer Letters
|
November 16, 2010
Genomic instability and cancer: lessons learned from human papillomaviruses
Nina Korzeniewski, Nicole Spardy, Anette Duensing, et al.
Molecular Biology of the Cell
|
September 24, 2010
Daughter centriole elongation is controlled by proteolysis
Nina Korzeniewski, Rolando Cuevas, Anette Duensing, et al.
Oncogene
|
February 3, 2004
The helix-loop-helix protein ID1 localizes to centrosomes and rapidly induces abnormal centrosome numbers
Jens Hasskarl, Stefan Duensing, Edwin Manuel, et al.
Urologic Oncology
|
October 22, 2013
Human papillomaviruses in urological malignancies: a critical assessment
Yanis Tolstov, Boris Hadaschik, Sascha Pahernik, et al.
Molecular Cancer
|
September 23, 2003
Excessive centrosome abnormalities without ongoing numerical chromosome instability in a Burkitt's lymphoma
Stefan Duensing, Benjamin H Lee, Paola Dal Cin, et al.
Urologic Oncology
|
June 17, 2014
High-risk prostate cancer: a disease of genomic instability
María A Tapia-Laliena, Nina Korzeniewski, Markus Hohenfellner, et al.
Page
of 14