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European Journal of Immunology|July 8, 2015
AIDing cancer treatment: Reducing AID activity via HSP90 inhibitionStefan Rebhandl, Roland Geisberger
Oncoscience|June 23, 2015
AID/APOBEC deaminases and cancerStefan Rebhandl, Michael Huemer, Richard Greil, et al.
European Journal of Immunology|March 27, 2014
Alternative splice variants of AID are not stoichiometrically present at the protein level in chronic lymphocytic leukemiaStefan Rebhandl, Michael Huemer, Nadja Zaborsky, et al.
European Journal of Immunology|September 3, 2014
AID induces intraclonal diversity and genomic damage in CD86(+) chronic lymphocytic leukemia cellsMichael Huemer, Stefan Rebhandl, Nadja Zaborsky, et al.
Molecular Cancer Research : MCR|December 10, 2017
Imprecision and DNA Break Repair Biased towards Incompatible End Joining in LeukemiaFranz Josef Gassner, Maria Schubert, Stefan Rebhandl, et al.
British Journal of Haematology|May 6, 2015
Chronic lymphocytic leukaemia induces an exhausted T cell phenotype in the TCL1 transgenic mouse modelFranz J Gassner, Nadja Zaborsky, Kemal Catakovic, et al.
Nucleic Acids Research|February 16, 2021
SAMHD1 restrains aberrant nucleotide insertions at repair junctions generated by DNA end joiningEkaterina Akimova, Franz Josef Gassner, Maria Schubert, et al.
Oncotarget|July 8, 2016
CD1d expression on chronic lymphocytic leukemia B cells affects disease progression and induces T cell skewing in CD8 positive and CD4CD8 double negative T cellsNadja Zaborsky, Franz Josef Gassner, Daniela Asslaber, et al.
Oncoimmunology|January 4, 2018
TIGIT expressing CD4+T cells represent a tumor-supportive T cell subset in chronic lymphocytic leukemiaKemal Catakovic, Franz Josef Gassner, Christoph Ratswohl, et al.
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