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Stephan Hinderlich

Showing results (41-50 of 52) with videos related to

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Bioengineering (Basel, Switzerland)|June 28, 2023
Seamless Coupling of Chemical Glycan Release and Labeling for an Accelerated Protein <i>N</i>-Glycan Sample Preparation WorkflowMumtaz Kasim, Anja Griebel, Grit Sandig, et al.
Plos One|June 19, 2008
UDP-N-acetylglucosamine 2-epimerase/N-acetylmannosamine kinase (GNE) binds to alpha-actinin 1: novel pathways in skeletal muscle?Shira Amsili, Hagit Zer, Stephan Hinderlich, et al.
Gene Regulation and Systems Biology|September 30, 2009
Preclinical assessment of wt GNE gene plasmid for management of hereditary inclusion body myopathy 2 (HIBM2)Chris Jay, Gregory Nemunaitis, John Nemunaitis, et al.
Chembiochem : a European Journal of Chemical Biology|March 28, 2017
Small Molecules Targeting Human N-Acetylmannosamine KinaseStephan Hinderlich, Martin Neuenschwander, Paul R Wratil, et al.
Biochemistry|March 1, 2006
Influence of UDP-GlcNAc 2-epimerase/ManNAc kinase mutant proteins on hereditary inclusion body myopathyJuliane Penner, Lars R Mantey, Sharona Elgavish, et al.
Bioengineering (Basel, Switzerland)|September 28, 2017
In Vitro Evaluation of Glycoengineered RSV-F in the Human Artificial Lymph Node ReactorLars Radke, Grit Sandig, Annika Lubitz, et al.
FEBS Letters|May 19, 2004
The homozygous M712T mutation of UDP-N-acetylglucosamine 2-epimerase/N-acetylmannosamine kinase results in reduced enzyme activities but not in altered overall cellular sialylation in hereditary inclusion body myopathyStephan Hinderlich, Ilan Salama, Iris Eisenberg, et al.
Chemical Science|April 3, 2023
Real-time monitoring of the sialic acid biosynthesis pathway by NMRJacob L Gorenflos López, Peter Schmieder, Kristin Kemnitz-Hassanin, et al.
Biochemical and Biophysical Research Communications|January 27, 2005
No overall hyposialylation in hereditary inclusion body myopathy myoblasts carrying the homozygous M712T GNE mutationIlan Salama, Stephan Hinderlich, Zipora Shlomai, et al.
Brain : a Journal of Neurology|June 18, 2010
Kelch-like homologue 9 mutation is associated with an early onset autosomal dominant distal myopathySebahattin Cirak, Florian von Deimling, Shrikesh Sachdev, et al.
Pageof 6

Showing results (41-50 of 52) with videos related to

Sort By:
Pageof 6
Bioengineering (Basel, Switzerland)|June 28, 2023
Seamless Coupling of Chemical Glycan Release and Labeling for an Accelerated Protein <i>N</i>-Glycan Sample Preparation WorkflowMumtaz Kasim, Anja Griebel, Grit Sandig, et al.
Plos One|June 19, 2008
UDP-N-acetylglucosamine 2-epimerase/N-acetylmannosamine kinase (GNE) binds to alpha-actinin 1: novel pathways in skeletal muscle?Shira Amsili, Hagit Zer, Stephan Hinderlich, et al.
Gene Regulation and Systems Biology|September 30, 2009
Preclinical assessment of wt GNE gene plasmid for management of hereditary inclusion body myopathy 2 (HIBM2)Chris Jay, Gregory Nemunaitis, John Nemunaitis, et al.
Chembiochem : a European Journal of Chemical Biology|March 28, 2017
Small Molecules Targeting Human N-Acetylmannosamine KinaseStephan Hinderlich, Martin Neuenschwander, Paul R Wratil, et al.
Biochemistry|March 1, 2006
Influence of UDP-GlcNAc 2-epimerase/ManNAc kinase mutant proteins on hereditary inclusion body myopathyJuliane Penner, Lars R Mantey, Sharona Elgavish, et al.
Bioengineering (Basel, Switzerland)|September 28, 2017
In Vitro Evaluation of Glycoengineered RSV-F in the Human Artificial Lymph Node ReactorLars Radke, Grit Sandig, Annika Lubitz, et al.
FEBS Letters|May 19, 2004
The homozygous M712T mutation of UDP-N-acetylglucosamine 2-epimerase/N-acetylmannosamine kinase results in reduced enzyme activities but not in altered overall cellular sialylation in hereditary inclusion body myopathyStephan Hinderlich, Ilan Salama, Iris Eisenberg, et al.
Chemical Science|April 3, 2023
Real-time monitoring of the sialic acid biosynthesis pathway by NMRJacob L Gorenflos López, Peter Schmieder, Kristin Kemnitz-Hassanin, et al.
Biochemical and Biophysical Research Communications|January 27, 2005
No overall hyposialylation in hereditary inclusion body myopathy myoblasts carrying the homozygous M712T GNE mutationIlan Salama, Stephan Hinderlich, Zipora Shlomai, et al.
Brain : a Journal of Neurology|June 18, 2010
Kelch-like homologue 9 mutation is associated with an early onset autosomal dominant distal myopathySebahattin Cirak, Florian von Deimling, Shrikesh Sachdev, et al.
Pageof 6