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Journal of Virology|March 27, 2015
Influenza virus-induced caspase-dependent enlargement of nuclear pores promotes nuclear export of viral ribonucleoprotein complexesDirk Mühlbauer, Julia Dzieciolowski, Martin Hardt, et al.The Journal of Experimental Medicine|December 10, 2008
Lung epithelial apoptosis in influenza virus pneumonia: the role of macrophage-expressed TNF-related apoptosis-inducing ligandSusanne Herold, Mirko Steinmueller, Werner von Wulffen, et al.Journal of Virology|December 17, 2009
The NS segment of an H5N1 highly pathogenic avian influenza virus (HPAIV) is sufficient to alter replication efficiency, cell tropism, and host range of an H7N1 HPAIVWenjun Ma, Dominique Brenner, Zhongfang Wang, et al.Systems Medicine (New Rochelle, N.Y.)|May 24, 2019
Time-Resolved Systems Medicine Reveals Viral Infection-Modulating Host TargetsChristian Wiwie, Irina Kuznetsova, Ahmed Mostafa, et al.Pathogens (Basel, Switzerland)|November 10, 2019
Evolution of H5-Type Avian Influenza A Virus Towards Mammalian Tropism in Egypt, 2014 to 2015Sara Hussein Mahmoud, Ahmed Mostafa, Rabeh El-Shesheny, et al.Viruses|March 26, 2022
Rocaglates as Antivirals: Comparing the Effects on Viral Resistance, Anti-Coronaviral Activity, RNA-Clamping on eIF4A and Immune Cell ToxicityWiebke Obermann, Alexandra Friedrich, Ramakanth Madhugiri, et al.Journal of Virology|April 19, 2019
Phosphoproteome Analysis of Cells Infected with Adapted and Nonadapted Influenza A Virus Reveals Novel Pro- and Antiviral Signaling NetworksAxel Weber, Sharmistha Dam, Vera V Saul, et al.Plos Pathogens|June 16, 2016
Correction: Macrophage-expressed IFN-β Contributes to Apoptotic Alveolar Epithelial Cell Injury in Severe Influenza Virus PneumoniaKatrin Högner, Thorsten Wolff, Stephan Pleschka, et al.Plos Pathogens|March 8, 2013
Macrophage-expressed IFN-β contributes to apoptotic alveolar epithelial cell injury in severe influenza virus pneumoniaKatrin Högner, Thorsten Wolff, Stephan Pleschka, et al.Virus Evolution|December 21, 2020
Genetic incompatibilities and reduced transmission in chickens may limit the evolution of reassortants between H9N2 and panzootic H5N8 clade 2.3.4.4 avian influenza virus showing high virulence for mammalsAhmed Mostafa, Claudia Blaurock, David Scheibner, et al.Pageof 10