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Haematologica|August 2, 2007
Clonal evolution in chronic lymphocytic leukemia: acquisition of high-risk genomic aberrations associated with unmutated VH, resistance to therapy, and short survivalStephan Stilgenbauer, Sandrine Sander, Lars Bullinger, et al.Blood|December 3, 2005
Distinct gene expression patterns in chronic lymphocytic leukemia defined by usage of specific VH genesDirk Kienle, Axel Benner, Alexander Kröber, et al.Proceedings of the National Academy of Sciences of the United States of America|May 11, 2006
Allelic silencing at the tumor-suppressor locus 13q14.3 suggests an epigenetic tumor-suppressor mechanismDaniel Mertens, Stephan Wolf, Cordula Tschuch, et al.British Journal of Haematology|July 9, 2003
Value of comparative genomic hybridization and fluorescence in situ hybridization for molecular diagnostics in multiple myelomaPeter Liebisch, Andreas Viardot, Nicole Bassermann, et al.Hematology. American Society of Hematology. Education Program|December 6, 2015
Prognostic markers and standard management of chronic lymphocytic leukemiaStephan StilgenbauerInternational Journal of Cancer|May 16, 2014
Loss of cooperativity of secreted CD40L and increased dose-response to IL4 on CLL cell viability correlates with enhanced activation of NF-kB and STAT6Nupur Bhattacharya, Michaela Reichenzeller, Maiwen Caudron-Herger, et al.British Journal of Haematology|May 28, 2011
Nurse-like cells show deregulated expression of genes involved in immunocompetenceNupur Bhattacharya, Susanne Diener, Irina S Idler, et al.Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|May 4, 2005
Evidence for distinct pathomechanisms in genetic subgroups of chronic lymphocytic leukemia revealed by quantitative expression analysis of cell cycle, activation, and apoptosis-associated genesDirk L Kienle, Christian Korz, Beate Hosch, et al.British Journal of Haematology|January 16, 2007
Reduced-intensity conditioning followed by T-cell depleted allogeneic stem cell transplantation for patients with chronic myeloid leukaemia and minimal residual disease at the time of transplant: high risk of molecular relapseMark Ringhoffer, Stephanie von Harsdorf, Michael Schmitt, et al.Pageof 70