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The Journal of Experimental Medicine|December 24, 2019
Innate immune priming in the absence of TAK1 drives RIPK1 kinase activity-independent pyroptosis, apoptosis, necroptosis, and inflammatory diseaseR K Subbarao Malireddi, Prajwal Gurung, Sannula Kesavardhana, et al.
Immunity|November 1, 2019
Amino Acids License Kinase mTORC1 Activity and Treg Cell Function via Small G Proteins Rag and RhebHao Shi, Nicole M Chapman, Jing Wen, et al.
Cell Reports|June 8, 2024
The interaction between RIPK1 and FADD controls perinatal lethality and inflammationDiego A Rodriguez, Bart Tummers, Jeremy J P Shaw, et al.
Life Science Alliance|March 11, 2024
Genetic ablation of <i>Immt</i> induces a lethal disruption of the MICOS complexStephanie M Rockfield, Meghan E Turnis, Ricardo Rodriguez-Enriquez, et al.
Nature|December 13, 2016
NLRC3 is an inhibitory sensor of PI3K-mTOR pathways in cancerRajendra Karki, Si Ming Man, R K Subbarao Malireddi, et al.
Diabetes|May 22, 2016
CRISPR-Cas9-Mediated Modification of the NOD Mouse Genome With Ptpn22R619W Mutation Increases Autoimmune DiabetesXiaotian Lin, Stephane Pelletier, Sebastien Gingras, et al.
Immunity|May 20, 2020
Caspase-8-Dependent Inflammatory Responses Are Controlled by Its Adaptor, FADD, and NecroptosisBart Tummers, Luigi Mari, Clifford S Guy, et al.
Cancer Research|July 24, 2021
Neuroblastoma Formation Requires Unconventional CD4 T Cells and Arginase-1-Dependent Myeloid CellsLee-Ann Van de Velde, E Kaitlynn Allen, Jeremy Chase Crawford, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 3, 2022
Caspase-8 and FADD prevent spontaneous ZBP1 expression and necroptosisDiego A Rodriguez, Giovanni Quarato, Swantje Liedmann, et al.
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