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Proceedings of the National Academy of Sciences of the United States of America|June 5, 2019
Ratiometric two-photon microscopy reveals attomolar copper buffering in normal and Menkes mutant cellsM Thomas Morgan, Daisy Bourassa, Shefali Harankhedkar, et al.
Molecular Biology of the Cell|December 26, 2008
SPE-39 family proteins interact with the HOPS complex and function in lysosomal deliveryGuang-dan Zhu, Gloria Salazar, Stephanie A Zlatic, et al.
Human Molecular Genetics|July 23, 2015
Neuronal copper homeostasis susceptibility by genetic defects in dysbindin, a schizophrenia susceptibility factorAvanti Gokhale, Alysia Vrailas-Mortimer, Jennifer Larimore, et al.
Frontiers in Genetics|March 28, 2017
Dysbindin Deficiency Modifies the Expression of GABA Neuron and Ion Permeation Transcripts in the Developing HippocampusJennifer Larimore, Stephanie A Zlatic, Miranda Arnold, et al.
Molecular Biology of the Cell|October 15, 2011
The schizophrenia susceptibility factor dysbindin and its associated complex sort cargoes from cell bodies to the synapseJennifer Larimore, Karine Tornieri, Pearl V Ryder, et al.
Eneuro|July 27, 2021
Heterogeneous Expression of Nuclear Encoded Mitochondrial Genes Distinguishes Inhibitory and Excitatory NeuronsMeghan E Wynne, Alicia R Lane, Kaela S Singleton, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 15, 2015
The N-ethylmaleimide-sensitive factor and dysbindin interact to modulate synaptic plasticityAvanti Gokhale, Ariana P Mullin, Stephanie A Zlatic, et al.
Biorxiv : the Preprint Server for Biology|February 6, 2026
Distinct signaling mechanisms and proteome phenotypes are elicited by compartment-specific genetic defects of copper homeostasisAlicia R Lane, Nadia Gonzalez, Avanti Gokhale, et al.
Molecular Biology of the Cell|May 13, 2026
Distinct signaling mechanisms and proteome phenotypes are elicited by compartment-specific genetic defects of copper homeostasisAlicia R Lane, Nadia Gonzalez, Avanti Gokhale, et al.
Cell Systems|February 5, 2018
Rare Disease Mechanisms Identified by Genealogical Proteomics of Copper Homeostasis Mutant PedigreesStephanie A Zlatic, Alysia Vrailas-Mortimer, Avanti Gokhale, et al.
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