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Stephanie Denk

Showing results (1-10 of 21) with videos related to

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Frontiers in Immunology|February 2, 2017
Complement in Pancreatic Disease-Perpetrator or Savior?Lucas Bettac, Stephanie Denk, Thomas Seufferlein, et al.
Advances in Experimental Medicine and Biology|September 28, 2011
Granzyme B: a new crossroad of complement and apoptosisMario Perl, Stephanie Denk, Miriam Kalbitz, et al.
Seminars in Immunology|May 10, 2016
Complement therapeutic strategies in trauma, hemorrhagic shock and systemic inflammation - closing Pandora's box?Markus Huber-Lang, Florian Gebhard, Christoph Q Schmidt, et al.
Plos One|February 4, 2014
Comparative analysis reveals distinct and overlapping functions of Mef2c and Mef2d during cardiogenesis in Xenopus laevisYanchun Guo, Susanne J Kühl, Astrid S Pfister, et al.
The Journal of Trauma and Acute Care Surgery|January 29, 2013
Inflammatory and apoptotic alterations in serum and injured tissue after experimental polytrauma in mice: distinct early response compared with single trauma or "double-hit" injurySebastian Weckbach, Christoph Hohmann, Sonja Braumueller, et al.
Journal of Immunology (Baltimore, Md. : 1950)|February 7, 2012
Factor VII-activating protease is activated in multiple trauma patients and generates anaphylatoxin C5aSandip M Kanse, Andrea Gallenmueller, Sacha Zeerleder, et al.
The Journal of Trauma and Acute Care Surgery|February 22, 2013
Apoptotic and inflammatory signaling via Fas and tumor necrosis factor receptor I contribute to the development of chest trauma-induced septic acute lung injurySebastian Weckbach, Christoph Hohmann, Stephanie Denk, et al.
Molecular Immunology|January 17, 2012
Cathepsin D is released after severe tissue trauma in vivo and is capable of generating C5a in vitroMarkus Huber-Lang, Stephanie Denk, Simone Fulda, et al.
Shock (Augusta, Ga.)|December 19, 2012
Silencing of fas, fas-associated via death domain, or caspase 3 differentially affects lung inflammation, apoptosis, and development of trauma-induced septic acute lung injuryMirko Philipp Messer, Philipp Kellermann, Sascha Jörn Weber, et al.
European Journal of Medical Research|August 26, 2015
The molecular fingerprint of lung inflammation after blunt chest traumaChristian Ehrnthaller, Michael Flierl, Mario Perl, et al.
Pageof 3

Showing results (1-10 of 21) with videos related to

Sort By:
Pageof 3
Frontiers in Immunology|February 2, 2017
Complement in Pancreatic Disease-Perpetrator or Savior?Lucas Bettac, Stephanie Denk, Thomas Seufferlein, et al.
Advances in Experimental Medicine and Biology|September 28, 2011
Granzyme B: a new crossroad of complement and apoptosisMario Perl, Stephanie Denk, Miriam Kalbitz, et al.
Seminars in Immunology|May 10, 2016
Complement therapeutic strategies in trauma, hemorrhagic shock and systemic inflammation - closing Pandora's box?Markus Huber-Lang, Florian Gebhard, Christoph Q Schmidt, et al.
Plos One|February 4, 2014
Comparative analysis reveals distinct and overlapping functions of Mef2c and Mef2d during cardiogenesis in Xenopus laevisYanchun Guo, Susanne J Kühl, Astrid S Pfister, et al.
The Journal of Trauma and Acute Care Surgery|January 29, 2013
Inflammatory and apoptotic alterations in serum and injured tissue after experimental polytrauma in mice: distinct early response compared with single trauma or "double-hit" injurySebastian Weckbach, Christoph Hohmann, Sonja Braumueller, et al.
Journal of Immunology (Baltimore, Md. : 1950)|February 7, 2012
Factor VII-activating protease is activated in multiple trauma patients and generates anaphylatoxin C5aSandip M Kanse, Andrea Gallenmueller, Sacha Zeerleder, et al.
The Journal of Trauma and Acute Care Surgery|February 22, 2013
Apoptotic and inflammatory signaling via Fas and tumor necrosis factor receptor I contribute to the development of chest trauma-induced septic acute lung injurySebastian Weckbach, Christoph Hohmann, Stephanie Denk, et al.
Molecular Immunology|January 17, 2012
Cathepsin D is released after severe tissue trauma in vivo and is capable of generating C5a in vitroMarkus Huber-Lang, Stephanie Denk, Simone Fulda, et al.
Shock (Augusta, Ga.)|December 19, 2012
Silencing of fas, fas-associated via death domain, or caspase 3 differentially affects lung inflammation, apoptosis, and development of trauma-induced septic acute lung injuryMirko Philipp Messer, Philipp Kellermann, Sascha Jörn Weber, et al.
European Journal of Medical Research|August 26, 2015
The molecular fingerprint of lung inflammation after blunt chest traumaChristian Ehrnthaller, Michael Flierl, Mario Perl, et al.
Pageof 3