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Human Molecular Genetics|August 20, 2019
Genetic variants in TRPM7 associated with unexplained stillbirth modify ion channel functionJames H Cartwright, Qadeer Aziz, Stephen C Harmer, et al.
Journal of Molecular and Cellular Cardiology|November 25, 2021
Inducing Ito,f and phase 1 repolarization of the cardiac action potential with a Kv4.3/KChIP2.1 bicistronic transgeneNan Wang, Eef Dries, Ewan D Fowler, et al.
Biochemical and Biophysical Research Communications|April 24, 2020
Serine mutation of a conserved threonine in the hERG K+ channel S6-pore region leads to loss-of-function through trafficking impairmentEhab Al-Moubarak, Yihong Zhang, Christopher E Dempsey, et al.
The Biochemical Journal|June 11, 2014
Cellular mechanisms underlying the increased disease severity seen for patients with long QT syndrome caused by compound mutations in KCNQ1Stephen C Harmer, Jagdeep S Mohal, Alice A Royal, et al.
Biochemical Pharmacology|August 23, 2025
Noncanonical pharmacological inhibition of the hERG K+ channel by a synthetic cannabinoidHongwei Cheng, Catherine Upex, Chunyun Du, et al.
Interface Focus|December 18, 2023
Evaluating pro-arrhythmogenic effects of the T634S-hERG mutation: insights from a simulation studyWei Hu, Wenfeng Zhang, Kevin Zhang, et al.
Frontiers in Cardiovascular Medicine|May 21, 2020
COVID-19 Management and Arrhythmia: Risks and Challenges for Clinicians Treating Patients Affected by SARS-CoV-2Alexander Carpenter, Owen J Chambers, Aziza El Harchi, et al.
Cellular and Molecular Life Sciences : CMLS|November 2, 2021
Inhibition of the hERG potassium channel by phenanthrene: a polycyclic aromatic hydrocarbon pollutantEhab Al-Moubarak, Holly A Shiels, Yihong Zhang, et al.
Frontiers in Physiology|April 11, 2019
Investigating the Complex Arrhythmic Phenotype Caused by the Gain-of-Function Mutation KCNQ1-G229DXin Zhou, Alfonso Bueno-Orovio, Richard J Schilling, et al.
Stem Cell Research|November 25, 2017
Ajmaline blocks INa and IKr without eliciting differences between Brugada syndrome patient and control human pluripotent stem cell-derived cardiac clustersDuncan C Miller, Stephen C Harmer, Ariel Poliandri, et al.
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