Search research articles
Contact Us
Filters
Showing results (31-40 of 44) with videos related to
Page
of 5
Sort By:
The Journal of Physiology
|
July 13, 2014
Complete reversal of Lambert-Eaton myasthenic syndrome synaptic impairment by the combined use of a K+ channel blocker and a Ca2+ channel agonist
Tyler B Tarr, David Lacomis, Stephen W Reddel, et al.
Frontiers in Cellular Neuroscience
|
July 21, 2025
Chronic benzodiazepine treatment triggers gephyrin scaffold destabilization and GABA<sub>A</sub>R subsynaptic reorganization
Caitlyn A Chapman, Nadya Povysheva, Tyler B Tarr, et al.
Human Molecular Genetics
|
February 9, 2023
Potentiation of neuromuscular transmission by a small molecule calcium channel gating modifier improves motor function in a severe spinal muscular atrophy mouse model
Kristine S Ojala, Cassandra J Kaufhold, Mykenzie R Davey, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
June 21, 2013
Evaluation of a novel calcium channel agonist for therapeutic potential in Lambert-Eaton myasthenic syndrome
Tyler B Tarr, Waqas Malick, Mary Liang, et al.
Annals of the New York Academy of Sciences
|
November 11, 2017
Lambert-Eaton myasthenic syndrome: mouse passive-transfer model illuminates disease pathology and facilitates testing therapeutic leads
Stephen D Meriney, Tyler B Tarr, Kristine S Ojala, et al.
Journal of Neurophysiology
|
April 19, 2023
Simulations of active zone structure and function at mammalian NMJs predict that loss of calcium channels alone is not sufficient to replicate LEMS effects
Scott P Ginebaugh, Yomna Badawi, Rozita Laghaei, et al.
Journal of Cell Science
|
March 11, 2010
Sustained synaptic-vesicle recycling by bulk endocytosis contributes to the maintenance of high-rate neurotransmitter release stimulated by glycerotoxin
Frederic A Meunier, Tam H Nguyen, Cesare Colasante, et al.
Journal of Neurophysiology
|
January 24, 2018
Transmitter release site organization can predict synaptic function at the neuromuscular junction
Rozita Laghaei, Jun Ma, Tyler B Tarr, et al.
American Journal of Human Genetics
|
June 12, 2010
GJC2 missense mutations cause human lymphedema
Robert E Ferrell, Catherine J Baty, Mark A Kimak, et al.
Scientific Reports
|
August 10, 2024
Neuromuscular dysfunction and pathogenesis in triosephosphate isomerase deficiency
Tracey D Myers, Yizhi Li, Stefanie Taiclet, et al.
Page
of 5
Search research articles
Search
Showing results (31-40 of 44) with videos related to
Sort By:
Page
of 5
The Journal of Physiology
|
July 13, 2014
Complete reversal of Lambert-Eaton myasthenic syndrome synaptic impairment by the combined use of a K+ channel blocker and a Ca2+ channel agonist
Tyler B Tarr, David Lacomis, Stephen W Reddel, et al.
Frontiers in Cellular Neuroscience
|
July 21, 2025
Chronic benzodiazepine treatment triggers gephyrin scaffold destabilization and GABA<sub>A</sub>R subsynaptic reorganization
Caitlyn A Chapman, Nadya Povysheva, Tyler B Tarr, et al.
Human Molecular Genetics
|
February 9, 2023
Potentiation of neuromuscular transmission by a small molecule calcium channel gating modifier improves motor function in a severe spinal muscular atrophy mouse model
Kristine S Ojala, Cassandra J Kaufhold, Mykenzie R Davey, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
June 21, 2013
Evaluation of a novel calcium channel agonist for therapeutic potential in Lambert-Eaton myasthenic syndrome
Tyler B Tarr, Waqas Malick, Mary Liang, et al.
Annals of the New York Academy of Sciences
|
November 11, 2017
Lambert-Eaton myasthenic syndrome: mouse passive-transfer model illuminates disease pathology and facilitates testing therapeutic leads
Stephen D Meriney, Tyler B Tarr, Kristine S Ojala, et al.
Journal of Neurophysiology
|
April 19, 2023
Simulations of active zone structure and function at mammalian NMJs predict that loss of calcium channels alone is not sufficient to replicate LEMS effects
Scott P Ginebaugh, Yomna Badawi, Rozita Laghaei, et al.
Journal of Cell Science
|
March 11, 2010
Sustained synaptic-vesicle recycling by bulk endocytosis contributes to the maintenance of high-rate neurotransmitter release stimulated by glycerotoxin
Frederic A Meunier, Tam H Nguyen, Cesare Colasante, et al.
Journal of Neurophysiology
|
January 24, 2018
Transmitter release site organization can predict synaptic function at the neuromuscular junction
Rozita Laghaei, Jun Ma, Tyler B Tarr, et al.
American Journal of Human Genetics
|
June 12, 2010
GJC2 missense mutations cause human lymphedema
Robert E Ferrell, Catherine J Baty, Mark A Kimak, et al.
Scientific Reports
|
August 10, 2024
Neuromuscular dysfunction and pathogenesis in triosephosphate isomerase deficiency
Tracey D Myers, Yizhi Li, Stefanie Taiclet, et al.
Page
of 5