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Archives of Neurology
|
November 11, 2009
Cellular prion protein mediates the toxicity of beta-amyloid oligomers: implications for Alzheimer disease
Haakon B Nygaard, Stephen M Strittmatter
Advances in Pharmacology (San Diego, Calif.)
|
February 8, 2018
Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5
A Harrison Brody, Stephen M Strittmatter
Journal of Molecular Medicine (Berlin, Germany)
|
December 5, 2009
Beta-amyloid oligomers and cellular prion protein in Alzheimer's disease
Erik C Gunther, Stephen M Strittmatter
Trends in Neurosciences
|
April 12, 2003
The Nogo-66 receptor: focusing myelin inhibition of axon regeneration
Aaron W McGee, Stephen M Strittmatter
Genome Biology
|
January 8, 2008
The reticulons: a family of proteins with diverse functions
Yvonne S Yang, Stephen M Strittmatter
Experimental Neurology
|
April 9, 2008
Functional MRI and other non-invasive imaging technologies: providing visual biomarkers for spinal cord structure and function after injury
Noam Y Harel, Stephen M Strittmatter
Current Alzheimer Research
|
January 29, 2008
Nogo receptor interacts with brain APP and Abeta to reduce pathologic changes in Alzheimer's transgenic mice
James H Park, Stephen M Strittmatter
Prion
|
September 19, 2012
Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease
Ji Won Um, Stephen M Strittmatter
Nature Reviews. Neuroscience
|
July 22, 2006
Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury?
Noam Y Harel, Stephen M Strittmatter
Results and Problems in Cell Differentiation
|
July 8, 2009
Axon regeneration in the peripheral and central nervous systems
Eric A Huebner, Stephen M Strittmatter
Page
of 20
Search research articles
Search
Showing results (11-20 of 194) with videos related to
Sort By:
Page
of 20
Archives of Neurology
|
November 11, 2009
Cellular prion protein mediates the toxicity of beta-amyloid oligomers: implications for Alzheimer disease
Haakon B Nygaard, Stephen M Strittmatter
Advances in Pharmacology (San Diego, Calif.)
|
February 8, 2018
Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5
A Harrison Brody, Stephen M Strittmatter
Journal of Molecular Medicine (Berlin, Germany)
|
December 5, 2009
Beta-amyloid oligomers and cellular prion protein in Alzheimer's disease
Erik C Gunther, Stephen M Strittmatter
Trends in Neurosciences
|
April 12, 2003
The Nogo-66 receptor: focusing myelin inhibition of axon regeneration
Aaron W McGee, Stephen M Strittmatter
Genome Biology
|
January 8, 2008
The reticulons: a family of proteins with diverse functions
Yvonne S Yang, Stephen M Strittmatter
Experimental Neurology
|
April 9, 2008
Functional MRI and other non-invasive imaging technologies: providing visual biomarkers for spinal cord structure and function after injury
Noam Y Harel, Stephen M Strittmatter
Current Alzheimer Research
|
January 29, 2008
Nogo receptor interacts with brain APP and Abeta to reduce pathologic changes in Alzheimer's transgenic mice
James H Park, Stephen M Strittmatter
Prion
|
September 19, 2012
Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease
Ji Won Um, Stephen M Strittmatter
Nature Reviews. Neuroscience
|
July 22, 2006
Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury?
Noam Y Harel, Stephen M Strittmatter
Results and Problems in Cell Differentiation
|
July 8, 2009
Axon regeneration in the peripheral and central nervous systems
Eric A Huebner, Stephen M Strittmatter
Page
of 20