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American Journal of Physiology. Heart and Circulatory Physiology|May 22, 2007
Ischemic preconditioning prevents in vivo hyperoxygenation in postischemic myocardium with preservation of mitochondrial oxygen consumptionXuehai Zhu, Bin Liu, Shaotang Zhou, et al.Free Radical Biology & Medicine|May 29, 2012
Protein thiyl radical mediates S-glutathionylation of complex IPatrick T Kang, Liwen Zhang, Chwen-Lih Chen, et al.American Journal of Physiology. Cell Physiology|October 6, 2006
Shear-induced reactive nitrogen species inhibit mitochondrial respiratory complex activities in cultured vascular endothelial cellsZhaosheng Han, Yeong-Renn Chen, Charles I Jones, et al.Free Radical Biology & Medicine|October 13, 2004
Involvement of protein radical, protein aggregation, and effects on NO metabolism in the hypochlorite-mediated oxidation of mitochondrial cytochrome cYeong-Renn Chen, Chwen-Lih Chen, Xiaoping Liu, et al.Free Radical Biology & Medicine|August 6, 2003
EPR spectroscopy studies on the structural transition of nitrosyl hemoglobin in the arterial-venous cycle of DEANO-treated rats as it relates to the proposed nitrosyl hemoglobin/nitrosothiol hemoglobin exchangeAdrian R Jaszewski, Yang C Fann, Yeong Renn Chen, et al.Journal of Molecular and Cellular Cardiology|July 23, 2018
Mitochondrial complex I in the post-ischemic heart: reperfusion-mediated oxidative injury and protein cysteine sulfonationPatrick T Kang, Chwen-Lih Chen, Paul Lin, et al.Journal of the American Society for Mass Spectrometry|September 16, 2008
Mass spectrometry profiles superoxide-induced intramolecular disulfide in the FMN-binding subunit of mitochondrial Complex ILiwen Zhang, Hua Xu, Chwen-Lih Chen, et al.American Journal of Physiology. Heart and Circulatory Physiology|January 11, 2011
Endothelial NOS activity and myocardial oxygen metabolism define the salvageable ischemic time window for ischemic postconditioningMing Cai, Yuanjing Li, Yi Xu, et al.Molecular and Cellular Biochemistry|October 24, 2009
Opening of the mitoKATP channel and decoupling of mitochondrial complex II and III contribute to the suppression of myocardial reperfusion hyperoxygenationBin Liu, Xuehai Zhu, Chwen-Lih Chen, et al.American Journal of Physiology. Heart and Circulatory Physiology|November 23, 2006
Endothelial nitric oxide synthase (NOS3) knockout decreases NOS2 induction, limiting hyperoxygenation and conferring protection in the postischemic heartXue Zhao, Yeong-Renn Chen, Guanglong He, et al.Pageof 11