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Proceedings of the National Academy of Sciences of the United States of America|September 13, 2012
Compartmentalization of superoxide dismutase 1 (SOD1G93A) aggregates determines their toxicitySarah J Weisberg, Roman Lyakhovetsky, Ayelet-chen Werdiger, et al.
Acta Neuropathologica|April 25, 2012
Distinct TDP-43 pathology in ALS patients with ataxin 2 intermediate-length polyQ expansionsMichael P Hart, Johannes Brettschneider, Virginia M Y Lee, et al.
Bio-Protocol|September 20, 2021
Arginine-rich Peptides Can Actively Mediate Liquid-liquid Phase SeparationSteven Boeynaems, Mathias De Decker, Peter Tompa, et al.
Acta Neuropathologica|June 9, 2016
Inside out: the role of nucleocytoplasmic transport in ALS and FTLDSteven Boeynaems, Elke Bogaert, Philip Van Damme, et al.
Pediatric Research|January 24, 2004
Tie2-Cre-induced inactivation of a conditional mutant Nf1 allele in mouse results in a myeloproliferative disorder that models juvenile myelomonocytic leukemiaAaron D Gitler, Yi Kong, John K Choi, et al.
Plos Biology|May 5, 2011
Molecular determinants and genetic modifiers of aggregation and toxicity for the ALS disease protein FUS/TLSZhihui Sun, Zamia Diaz, Xiaodong Fang, et al.
The Journal of Biological Chemistry|May 26, 2009
TDP-43 is intrinsically aggregation-prone, and amyotrophic lateral sclerosis-linked mutations accelerate aggregation and increase toxicityBrian S Johnson, David Snead, Jonathan J Lee, et al.
Traffic (Copenhagen, Denmark)|December 10, 2015
Analysis of COPII Vesicles Indicates a Role for the Emp47-Ssp120 Complex in Transport of Cell Surface GlycoproteinsNeil G Margulis, Joshua D Wilson, Christine M Bentivoglio, et al.
Scientific Reports|February 13, 2016
Drosophila screen connects nuclear transport genes to DPR pathology in c9ALS/FTDSteven Boeynaems, Elke Bogaert, Emiel Michiels, et al.
Developmental Biology|May 11, 2005
Insertion of Cre into the Pax3 locus creates a new allele of Splotch and identifies unexpected Pax3 derivativesKurt A Engleka, Aaron D Gitler, Maozhen Zhang, et al.
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