Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Steven Holland

Showing results (21-30 of 28) with videos related to

Pageof 3
Sort By:
You have reached the last page of results.This site can display upto 28 results.
Blood|October 4, 2017
Effective "activated PI3Kδ syndrome"-targeted therapy with the PI3Kδ inhibitor leniolisibV Koneti Rao, Sharon Webster, Virgil A S H Dalm, et al.
Biorxiv : the Preprint Server for Biology|March 2, 2022
Genetically diverse mouse models of SARS-CoV-2 infection reproduce clinical variation in type I interferon and cytokine responses in COVID-19Shelly Robertson, Olivia Bedard, Kristin McNally, et al.
Blood|April 27, 2016
Heterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotypeJulie Toubiana, Satoshi Okada, Julia Hiller, et al.
Human Molecular Genetics|January 27, 2011
Partial recessive IFN-γR1 deficiency: genetic, immunological and clinical features of 14 patients from 11 kindredsIthaisa Sologuren, Stéphanie Boisson-Dupuis, Jose Pestano, et al.
Frontiers in Immunology|October 19, 2017
Corrigendum: Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56<sup>bright</sup> NKG2A<sup>+++</sup> Cells, and Yet Display Increased Degranulation and Higher Perforin ContentKerry Dobbs, Giovanna Tabellini, Enrica Calzoni, et al.
Frontiers in Immunology|August 4, 2017
Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56<sup>bright</sup> NKG2A<sup>+++</sup> Cells, and Yet Display Increased Degranulation and Higher Perforin ContentKerry Dobbs, Giovanna Tabellini, Enrica Calzoni, et al.
Medrxiv : the Preprint Server for Health Sciences|July 20, 2021
Genetic regulation of <i>OAS1</i> nonsense-mediated decay underlies association with risk of severe COVID-19A Rouf Banday, Megan L Stanifer, Oscar Florez-Vargas, et al.
Nature Genetics|July 14, 2022
Genetic regulation of OAS1 nonsense-mediated decay underlies association with COVID-19 hospitalization in patients of European and African ancestriesA Rouf Banday, Megan L Stanifer, Oscar Florez-Vargas, et al.
Pageof 3

Showing results (21-30 of 28) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 28 results.
Blood|October 4, 2017
Effective "activated PI3Kδ syndrome"-targeted therapy with the PI3Kδ inhibitor leniolisibV Koneti Rao, Sharon Webster, Virgil A S H Dalm, et al.
Biorxiv : the Preprint Server for Biology|March 2, 2022
Genetically diverse mouse models of SARS-CoV-2 infection reproduce clinical variation in type I interferon and cytokine responses in COVID-19Shelly Robertson, Olivia Bedard, Kristin McNally, et al.
Blood|April 27, 2016
Heterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotypeJulie Toubiana, Satoshi Okada, Julia Hiller, et al.
Human Molecular Genetics|January 27, 2011
Partial recessive IFN-γR1 deficiency: genetic, immunological and clinical features of 14 patients from 11 kindredsIthaisa Sologuren, Stéphanie Boisson-Dupuis, Jose Pestano, et al.
Frontiers in Immunology|October 19, 2017
Corrigendum: Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56<sup>bright</sup> NKG2A<sup>+++</sup> Cells, and Yet Display Increased Degranulation and Higher Perforin ContentKerry Dobbs, Giovanna Tabellini, Enrica Calzoni, et al.
Frontiers in Immunology|August 4, 2017
Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56<sup>bright</sup> NKG2A<sup>+++</sup> Cells, and Yet Display Increased Degranulation and Higher Perforin ContentKerry Dobbs, Giovanna Tabellini, Enrica Calzoni, et al.
Medrxiv : the Preprint Server for Health Sciences|July 20, 2021
Genetic regulation of <i>OAS1</i> nonsense-mediated decay underlies association with risk of severe COVID-19A Rouf Banday, Megan L Stanifer, Oscar Florez-Vargas, et al.
Nature Genetics|July 14, 2022
Genetic regulation of OAS1 nonsense-mediated decay underlies association with COVID-19 hospitalization in patients of European and African ancestriesA Rouf Banday, Megan L Stanifer, Oscar Florez-Vargas, et al.
Pageof 3