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Steven J Coultrap

Showing results (21-30 of 45) with videos related to

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Molecular Pharmacology|May 30, 2026
The CaMKII D135N mutation blocks kinase activity and reduces GluN2B bindingMatthew E Larsen, C Madison Barker, Raul Satoshi Vargas, et al.
Biorxiv : the Preprint Server for Biology|September 26, 2025
The CaMKII D135N mutation blocks kinase activity and reduces GluN2B bindingMatthew E Larsen, C Madison Barker, Raul Satoshi Vargas, et al.
Science Signaling|July 25, 2023
Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in miceNicole L Rumian, Ronald K Freund, Mark L Dell'Acqua, et al.
Cell Reports|December 29, 2021
Conserved and divergent features of neuronal CaMKII holoenzyme structure, function, and high-order assemblyOlivia R Buonarati, Adam P Miller, Steven J Coultrap, et al.
Brain Research. Molecular Brain Research|April 29, 2005
Differential expression of NMDA receptor subunits and splice variants among the CA1, CA3 and dentate gyrus of the adult ratSteven J Coultrap, Kristin M Nixon, Rachel M Alvestad, et al.
Brain Research|February 8, 2014
Excitotoxic glutamate insults block autophagic flux in hippocampal neuronsJacqueline R Kulbe, Jean M Mulcahy Levy, Steven J Coultrap, et al.
Iscience|May 27, 2022
Aβ-induced synaptic impairments require CaMKII activity that is stimulated by indirect signaling eventsCarolyn Nicole Brown, Nicole L Rumian, Jonathan E Tullis, et al.
The Journal of Biological Chemistry|January 22, 2003
Tyrosine dephosphorylation and ethanol inhibition of N-Methyl-D-aspartate receptor functionRachel M Alvestad, David R Grosshans, Steven J Coultrap, et al.
Nature Communications|May 26, 2018
Publisher Correction: The CaMKII holoenzyme structure in activation-competent conformationsJanette B Myers, Vincent Zaegel, Steven J Coultrap, et al.
Biorxiv : the Preprint Server for Biology|July 9, 2025
CaMKII monomers are sufficient for GluN2B binding, co-condensation, and synaptic potentiationCarolyn Nicole Brown, C Madison Barker, Carley N Miller, et al.
Pageof 5

Showing results (21-30 of 45) with videos related to

Sort By:
Pageof 5
Molecular Pharmacology|May 30, 2026
The CaMKII D135N mutation blocks kinase activity and reduces GluN2B bindingMatthew E Larsen, C Madison Barker, Raul Satoshi Vargas, et al.
Biorxiv : the Preprint Server for Biology|September 26, 2025
The CaMKII D135N mutation blocks kinase activity and reduces GluN2B bindingMatthew E Larsen, C Madison Barker, Raul Satoshi Vargas, et al.
Science Signaling|July 25, 2023
Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in miceNicole L Rumian, Ronald K Freund, Mark L Dell'Acqua, et al.
Cell Reports|December 29, 2021
Conserved and divergent features of neuronal CaMKII holoenzyme structure, function, and high-order assemblyOlivia R Buonarati, Adam P Miller, Steven J Coultrap, et al.
Brain Research. Molecular Brain Research|April 29, 2005
Differential expression of NMDA receptor subunits and splice variants among the CA1, CA3 and dentate gyrus of the adult ratSteven J Coultrap, Kristin M Nixon, Rachel M Alvestad, et al.
Brain Research|February 8, 2014
Excitotoxic glutamate insults block autophagic flux in hippocampal neuronsJacqueline R Kulbe, Jean M Mulcahy Levy, Steven J Coultrap, et al.
Iscience|May 27, 2022
Aβ-induced synaptic impairments require CaMKII activity that is stimulated by indirect signaling eventsCarolyn Nicole Brown, Nicole L Rumian, Jonathan E Tullis, et al.
The Journal of Biological Chemistry|January 22, 2003
Tyrosine dephosphorylation and ethanol inhibition of N-Methyl-D-aspartate receptor functionRachel M Alvestad, David R Grosshans, Steven J Coultrap, et al.
Nature Communications|May 26, 2018
Publisher Correction: The CaMKII holoenzyme structure in activation-competent conformationsJanette B Myers, Vincent Zaegel, Steven J Coultrap, et al.
Biorxiv : the Preprint Server for Biology|July 9, 2025
CaMKII monomers are sufficient for GluN2B binding, co-condensation, and synaptic potentiationCarolyn Nicole Brown, C Madison Barker, Carley N Miller, et al.
Pageof 5